Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Angelo H Cho"'
Autor:
Toufik Taib, Claire Leconte, Juliette Van Steenwinckel, Angelo H Cho, Bruno Palmier, Egle Torsello, Rene Lai Kuen, Somfieme Onyeomah, Karine Ecomard, Chiara Benedetto, Bérard Coqueran, Anne-Catherine Novak, Edwige Deou, Michel Plotkine, Pierre Gressens, Catherine Marchand-Leroux, Valérie C Besson
Publikováno v:
PLoS ONE, Vol 12, Iss 9, p e0184811 (2017)
Traumatic brain injury (TBI) results in white matter injury (WMI) that is associated with neurological deficits. Neuroinflammation originating from microglial activation may participate in WMI and associated disorders. To date, there is little inform
Externí odkaz:
https://doaj.org/article/153e34942e784f7d9057577e137eea93
Autor:
Pierre Gressens, Bérard Coqueran, Anne-Catherine Novak, Angelo H. Cho, Toufik Taib, Catherine Marchand-Leroux, Edwige Deou, Valérie C. Besson, Juliette Van Steenwinckel, Karine Ecomard, Michel Plotkine, Bruno Palmier, Claire Leconte, Egle Torsello, Somfieme Onyeomah, René Lai Kuen, Chiara Benedetto
Publikováno v:
PLoS ONE
PLoS ONE, Vol 12, Iss 9, p e0184811 (2017)
PLoS ONE, Vol 12, Iss 9, p e0184811 (2017)
Traumatic brain injury (TBI) results in white matter injury (WMI) that is associated with neurological deficits. Neuroinflammation originating from microglial activation may participate in WMI and associated disorders. To date, there is little inform
Autor:
Eleni Siopi, Mehrnaz Jafarian-Tehrani, Angelo H. Cho, Michel Plotkine, Nicole Croci, Catherine Marchand-Leroux, Shadi Homsi
Publikováno v:
Journal of Neurotrauma. 28:2135-2143
Traumatic brain injury (TBI) induces both focal and diffuse lesions that are concurrently responsible for the ensuing morbidity and mortality and for which no established treatment is available. It has been recently reported that an endogenous neurop
Autor:
Eleni Siopi, Sandra Vidal-Lletjós, Angelo H. Cho, Mehrnaz Jafarian-Tehrani, Catherine Marchand-Leroux, Gemma Llufriu-Dabén, Michel Plotkine
Publikováno v:
Neuropharmacology. 67
Traumatic brain injury (TBI) evokes an intense neuroinflammatory reaction that is essentially mediated by activated microglia and that has been reported to act as a secondary injury mechanism that further promotes neuronal death. It involves the exce