Zobrazeno 1 - 10
of 16
pro vyhledávání: '"Angelika Griep"'
Autor:
Nicole Schröder, Anja Schaffrath, Josua A. Welter, Tim Putzka, Angelika Griep, Patrick Ziegler, Elisa Brandt, Sebastian Samer, Michael T. Heneka, Hannes Kaddatz, Jiangshan Zhan, Eugenia Kipp, Thomas Pufe, Simone C. Tauber, Markus Kipp, Lars-Ove Brandenburg
Publikováno v:
Journal of Neuroinflammation, Vol 17, Iss 1, Pp 1-15 (2020)
Abstract Background An important hallmark of Alzheimer’s disease (AD) is the increase of Aβ1-42 burden and its accumulation to senile plaques, leading the reactive gliosis and neurodegeneration. The modulation of glia cell function represents an a
Externí odkaz:
https://doaj.org/article/b44e213a449e4510a06c4e89b88026b5
Autor:
Roisin M. McManus, Max P Komes, Angelika Griep, Francesco Santarelli, Stephanie Schwartz, Michelle‐Amirah Khalil, Mario Lauterbach, David Gomez‐Cabrero, Jesper Tegner, Karsten Hiller, Eicke Latz, Michael T. Heneka
Publikováno v:
Alzheimer's & Dementia. 18
Autor:
Sebastian Samer, Thomas Pufe, EF Brandt, Patrick Ziegler, Jiangshan Zhan, Hannes Kaddatz, Josua A. Welter, Eugenia Kipp, Nicole Schröder, Lars-Ove Brandenburg, Markus Kipp, Michael T. Heneka, Simone C. Tauber, Tim Putzka, Anja Schaffrath, Angelika Griep
Publikováno v:
Journal of Neuroinflammation, Vol 17, Iss 1, Pp 1-15 (2020)
Journal of Neuroinflammation
Journal of neuroinflammation 17(1), 131 (2020). doi:10.1186/s12974-020-01816-2
Journal of Neuroinflammation
Journal of neuroinflammation 17(1), 131 (2020). doi:10.1186/s12974-020-01816-2
Background An important hallmark of Alzheimer’s disease (AD) is the increase of Aβ1-42 burden and its accumulation to senile plaques, leading the reactive gliosis and neurodegeneration. The modulation of glia cell function represents an attractive
Autor:
Roisin McManus, Max P. Komes, Angelika Griep, Francesco Santarelli, Stephanie Schwartz, Juan Ramon Perea, Michelle-Amirah Khalil, Mario Lauterbach, Lea Heinemann, Titus Schlüter, Juan F. Rodriguez Alcazar, Susanne F. Schmidt, Jasper Spitzer, Peri Noori, Alberto Maillo, David Gomez-Cabrero, Jesper Tegnér, Karsten Hiller, Eicke Latz, Michael T. Heneka
Publikováno v:
SSRN Electronic Journal.
Autor:
Annett Halle, Matthias Brückner, Christiane Kummer, Marc Beyer, Heela Sarlus, Ana Vieira-Saecker, Stephanie Schwartz, Christina Ising, Michael T. Heneka, Eicke Latz, Joachim L. Schultze, Markus P. Kummer, Kristian Händler, Angelika Griep
Publikováno v:
The EMBO journal 40(24), e108662 (2021). doi:10.15252/embj.2021108662
The EMBO Journal
The EMBO Journal
Chronic neuroinflammation is a pathogenic component of Alzheimer’s disease (AD) that may limit the ability of the brain to clear amyloid deposits and cellular debris. Tight control of the immune system is therefore key to sustain the ability of the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::90cb05e330be04d3ebfcce0c4bc7c292
https://pub.dzne.de/record/163394
https://pub.dzne.de/record/163394
Publikováno v:
Alzheimer's & Dementia. 16
Autor:
David Blum, Douglas T. Golenbock, Frederic Brosseron, Shuangshuang Zhang, Dario Tejera, Shadi Albasset, Michael T. Heneka, Angelika Griep, Rakez Kayed, Susanne V. Schmidt, Róisín M. McManus, Sabine Opitz, James Stunden, Christina Ising, Stephanie Schwartz, Eicke Latz, Carmen Venegas, Luc Buée, Francesco Santarelli, Ana Vieira-Saecker, Hannah Scheiblich
Publikováno v:
Alzheimer's & Dementia. 16
Autor:
Michael T. Heneka, Hannah Scheiblich, Ronald Melki, Luc Bousset, Stephanie Schwartz, Angelika Griep, Eicke Latz
Publikováno v:
SSRN Electronic Journal.
Parkinson’s disease (PD) is the second most common age-related neurodegenerative disorder characterized by the formation of cellular inclusions inside neurons that are rich in an abnormal form of the protein α-synuclein (α-syn). Microglia are the
Autor:
Nicole Schröder, Anja Schaffrath, Josua A. Welter, Tim Putzka, Angelika Griep, Patrick Ziegler, Elisa Brandt, Sebastian Samer, Michael T. Heneka, Eugenia Kipp, Thomas Pufe, Simone C. Tauber, Markus Kipp, Lars-Ove Brandenburg
Background: An important hallmark of Alzheimer's disease (AD) is the increase of Aβ1-42 burden and its accumulation to senile plaques, leading the reactive gliosis and neurodegeneration. The modulation of glia cell function represent an attractive t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::71806d5294a4e88528370234313357bf
https://doi.org/10.21203/rs.2.17967/v1
https://doi.org/10.21203/rs.2.17967/v1
Autor:
Michael T. Heneka, Susanne Schmidt, Luc Bousset, Lena Wischhof, Alexandru Odainic, Daniele Bano, Stephanie Schwartz, Cira Dansokho, Jasper Spitzer, Ronald Melki, Hannah Scheiblich, Angelika Griep, Dilek Mercan, Eicke Latz, Frederik Eikens
Publikováno v:
Cell 184(20), 5089-5106.e21 (2021). doi:10.1016/j.cell.2021.09.007
Cell
Cell, 2021, 184, pp.5089-5106. ⟨10.1016/j.cell.2021.09.007⟩
Cell, Elsevier, 2021, 184, pp.5089-5106. ⟨10.1016/j.cell.2021.09.007⟩
Cell
Cell, 2021, 184, pp.5089-5106. ⟨10.1016/j.cell.2021.09.007⟩
Cell, Elsevier, 2021, 184, pp.5089-5106. ⟨10.1016/j.cell.2021.09.007⟩
International audience; Microglia are the CNS resident immune cells that react to misfolded proteins through pattern recognition receptor ligation and activation of inflammatory pathways. Here, we studied how microglia handle and cope with $\alpha$-s