Zobrazeno 1 - 10
of 14
pro vyhledávání: '"Angelika Brekman"'
Publikováno v:
Oncotarget
// Nandini Kundu 1, 2 , Angelika Brekman 1, 3 , Jun Yeob Kim 1 , Gu Xiao 1 , Chong Gao 1, 2 and Jill Bargonetti 1, 2, 3, 4 1 The Department of Biological Sciences Hunter College, City University of New York, New York, NY 10065, USA 2 PhD Program in B
Publikováno v:
The FASEB Journal. 34:1-1
Autor:
Bradley Witover, Ronald G. Crystal, IonWa Chao, Angelika Brekman, Wu-Lin Zuo, Tomoya Fukui, Renat Shaykhiev
Publikováno v:
Proceedings of the National Academy of Sciences. 110:12102-12107
The airway epithelium of smokers acquires pathological phenotypes, including basal cell (BC) and/or goblet cell hyperplasia, squamous metaplasia, structural and functional abnormalities of ciliated cells, decreased number of secretoglobin (SCGB1A1)-e
Autor:
Yael Strulovici-Barel, Rui Wang, Angelika Brekman, Jason G. Mezey, Haixia Zhou, Matthew S. Walters, Jacqueline Salit, Robert J. Kaner, Guoqing Wang, Wu-Lin Zuo, Renat Shaykhiev, Michelle R. Staudt, Xuemei Ou, Francisco Agosto-Perez, Ronald G. Crystal
Publikováno v:
Journal of immunology (Baltimore, Md. : 1950). 196(7)
In the process of seeking novel lung host defense regulators by analyzing genome-wide RNA sequence data from normal human airway epithelium, we detected expression of POU domain class 2–associating factor 1 (POU2AF1), a known transcription cofactor
Publikováno v:
American journal of respiratory cell and molecular biology. 51(5)
Airway epithelium ciliated cells play a central role in clearing the lung of inhaled pathogens and xenobiotics, and cilia length and coordinated beating are important for airway clearance. Based on in vivo studies showing that the airway epithelium o
Publikováno v:
A13. ADVANCES IN COPD PATHOGENESIS AND EXACERBATION SUSCEPTIBILITY.
Publikováno v:
Breast Cancer Research : BCR
Following publication of our article, the authors noticed that the data incorporated into Figure 4f was incorrect. The first column should be changed from 44, 13 and 46 to read 43, 13 and 44 instead. A corrected version of Figure 4f can be found over
Publikováno v:
Breast Cancer Research : BCR
Introduction Estrogen receptor positive breast cancers often have high levels of Mdm2. We investigated if estrogen signaling in such breast cancers occurred through an Mdm2 mediated pathway with subsequent inactivation of p53. Methods We examined the
Autor:
Nicoleta C, Arva, Kathryn E, Talbott, Danielle R, Okoro, Angelika, Brekman, Wei Gang, Qiu, Jill, Bargonetti
Publikováno v:
Ethnicitydisease. 18(2 Suppl 2)
Mdm2 inhibits p53 transactivation by forming a p53-Mdm2 complex on chromatin. Upon DNA damage-induced complex disruption, such latent p53 can be activated, but in cells overexpressing Mdm2 because of a homozygous single nucleotide polymorphism at pos
Autor:
Akira Kawamura, Anna Takaoka, Clifford E. Soll, Stephanie Wolfe, Angelika Brekman, Tal H. Hasson, Yevgeniy A. Grigoryev
Publikováno v:
Bioorganicmedicinal chemistry letters. 16(11)
A screening methodology called 'genomic screening' was established to identify natural products that can regulate cellular gene expression. Application of genomic screening to Keishi-bukuryo-gan (KBG), a Japanese herbal medicine formulation, identifi