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pro vyhledávání: '"Angela D. Hoptak-Solga"'
Publikováno v:
FEBS letters. 581(17)
Mutations in the zebrafish connexin43 (cx43) gene cause the short fin phenotype, indicating that direct cell–cell communication contributes to bone length. Three independently generated cx43 alleles exhibit short segments of variable sizes, suggest
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fbee06d545ed1959d7213fac759f3f53
https://pubmed.ncbi.nlm.nih.gov/17599838
https://pubmed.ncbi.nlm.nih.gov/17599838
Publikováno v:
The FASEB Journal. 22:17-17
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::f899976ec24aa594e7109738d777a92b
https://doi.org/10.1096/fasebj.22.2_supplement.17
https://doi.org/10.1096/fasebj.22.2_supplement.17
Autor:
Angela D. Hoptak-Solga, Sarah Nielsen, Isha H. Jain, Ryan Thummel, M. Kathryn Iovine, David R. Hyde
Publikováno v:
Developmental Biology. (2):541-548
In zebrafish, mutations in the gap junction gene connexin43 lead to short bony fin ray segments that give rise to the short fin phenotype. The sofb123 mutant exhibits fins that are half the length of wild-type fins and have reduced levels of cx43 mRN
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4aff296bf6c1803193baed85391c5531