Zobrazeno 1 - 10
of 18
pro vyhledávání: '"Aneta Kaniak"'
Publikováno v:
PLoS ONE, Vol 12, Iss 7, p e0180153 (2017)
Rad27/FEN1 nuclease that plays important roles in the maintenance of DNA stability in the nucleus has recently been shown to reside in mitochondria. Accordingly, it has been established that Rad27 deficiency causes increased mutagenesis, but decrease
Externí odkaz:
https://doaj.org/article/56c6b264512646e598f3edccde05a1e5
Autor:
Joanna Kaminska, Damian Kolakowski, Weronika Rzepnikowska, Teresa Zoladek, Aneta Kaniak-Golik
Publikováno v:
International Journal of Molecular Sciences
Volume 22
Issue 22
International Journal of Molecular Sciences, Vol 22, Iss 12274, p 12274 (2021)
Volume 22
Issue 22
International Journal of Molecular Sciences, Vol 22, Iss 12274, p 12274 (2021)
VPS13 proteins are evolutionarily conserved. Mutations in the four human genes (VPS13A-D) encoding VPS13A-D proteins are linked to developmental or neurodegenerative diseases. The relationship between the specific localization of individual VPS13 pro
Autor:
Miroslaw Bik-Multanowski, Agnieszka Pollak, Ewa Sledziewska-Gojska, Aneta Kaniak-Golik, Jolanta Fijak-Moskal, Jarosław Poznański, Rafał Płoski, Victor Murcia Pienkowski, Renata Kuberska, Małgorzata Rydzanicz, Agnieszka Halas
REV3L encodes a catalytic subunit of DNA polymerase zeta (Pol zeta) which is essential for the tolerance of DNA damage by inducing translesion synthesis (TLS). So far, the only Mendelian disease associated with REV3L was Moebius syndrome (3 patients
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1b09a60cb62833baa58a52d5bb4aa7f2
https://ruj.uj.edu.pl/xmlui/handle/item/270271
https://ruj.uj.edu.pl/xmlui/handle/item/270271
Publikováno v:
FEMS Microbiology Reviews
Cells are constantly confronted with endogenous and exogenous factors that affect their genomes. Eons of evolution have allowed the cellular mechanisms responsible for preserving the genome to adjust for achieving contradictory objectives: to maintai
Autor:
Adrianna Skoneczna, Aneta Kaniak-Golik
Publikováno v:
Free Radical Biology and Medicine. 82:73-104
The proper functioning of the cell depends on preserving the cellular genome. In yeast cells, a limited number of genes are located on mitochondrial DNA. Although the mechanisms underlying nuclear genome maintenance are well understood, much less is
Publikováno v:
Biochimica et Biophysica Acta (BBA) - Bioenergetics. 1797:1086-1098
As a legacy of their endosymbiotic eubacterial origin, mitochondria possess a residual genome, encoding only a few proteins and dependent on a variety of factors encoded by the nuclear genome for its maintenance and expression. As a facultative anaer
Publikováno v:
Eukaryotic Cell. 3:221-231
The yeast Saccharomyces cerevisiae senses glucose, its preferred carbon source, through multiple signal transduction pathways. In one pathway, glucose represses the expression of many genes through the Mig1 transcriptional repressor, which is regulat
Publikováno v:
PLoS ONE, Vol 12, Iss 7, p e0180153 (2017)
PLoS ONE
PLoS ONE
Rad27/FEN1 nuclease that plays important roles in the maintenance of DNA stability in the nucleus has recently been shown to reside in mitochondria. Accordingly, it has been established that Rad27 deficiency causes increased mutagenesis, but decrease
Autor:
Piotr P. Slonimski, Agnès Baudin-Baillieu, Jean-Paul di Rago, Aneta Kaniak, Robert Gromadka, Gabriela Orlowska, Olga Groudinsky, Jean-Yves Coppee, Klaus-Jörg Rieger, Gordana Aljinovic
Publikováno v:
Yeast. 13:1547-1562
In 1993, a pilot project for the functional analysis of newly discovered open reading frames, presumably coding for proteins, from yeast chromosome III was launched by the European Community. In the frame of this programme, we have developed a large-
Autor:
J.-P. di Rago, Aneta Kaniak, Jean-Yves Coppee, Olga Groudinsky, Klaus-Jörg Rieger, Piotr P. Slonimski
Publikováno v:
Yeast. 12:577-582
In the frame of the European Pilot Project for the functional analysis of newly discovered open reading frames (ORFs) from Saccharomyces cerevisiae chromosome III, we have deleted entirely the YCR46C ORF by a one-step polymerase chain reaction method