Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Andrey E. Belykh"'
Publikováno v:
Research Results in Pharmacology, Vol 7, Iss 1, Pp 27-32 (2021)
Introduction: The His-Phe-Arg-Trp sequence corresponding to the 6-9th amino acid residue of the adrenocorticotropic hormone molecule (ACTH6-9) is the critical pharmacophore of all endogenous melanocortin receptor agonists. In order to effects prolong
Externí odkaz:
https://doaj.org/article/a22d9b578a4242eaa1414a907872c8c4
Autor:
Alexander A. Dolskiy, Andrey S. Gudymo, Oleg S. Taranov, Irina V. Grishchenko, Ekaterina M. Shitik, Dmitry Yu Prokopov, Vladislav O. Soldatov, Elvira V. Sobolevskaya, Sergey A. Bodnev, Natalia V. Danilchenko, Anastasia A. Moiseeva, Polina Y. Torzhkova, Yulia A. Bulanovich, Galina S. Onhonova, Elena K. Ivleva, Marina V. Kubekina, Andrey E. Belykh, Tatiana V. Tregubchak, Alexander B. Ryzhikov, Elena V. Gavrilova, Rinat A. Maksyutov, Alexey V. Deykin, Dmitry V. Yudkin
Publikováno v:
Frontiers in Molecular Biosciences, Vol 8 (2022)
The novel coronavirus disease COVID-19 has become one of the most socially significant infections. One of the main models for COVID-19 pathogenesis study and anti-COVID-19 drug development is laboratory animals sensitive to the virus. Herein, we repo
Externí odkaz:
https://doaj.org/article/0750c6fbe87940c5857a6725fe27bfc4
Autor:
Vladislav O. Soldatov, Alexey V. Deykin, Andrey E Belykh, Michail S. Kukharsky, Andrey M Sobolev
Publikováno v:
Eye and Brain
Amyotrophic lateral sclerosis (ALS) is a fatal neurodegenerative disease resulting in a gradual loss of motor neuron function. Although ophthalmic complaints are not presently considered a classic symptom of ALS, retinal changes such as thinning, axo
Autor:
Egor Churkin, Alexey Polonikov, Ekaterina Barysheva, M.S. Nazarenko, Vladimir P. Ivanov, Andrey E Belykh, Olga Bushueva, I.A. Koroleva, A.V. Markov
Publikováno v:
Journal of Molecular Neuroscience. 71:1914-1932
Dysregulation of the oxidant-antioxidant system contributes to the pathogenesis of cerebral stroke (CS). Epigenetic changes of redox homeostasis genes, such as glutamate-cysteine ligase (GCLM), glutathione-S-transferase-P1 (GSTP1), thioredoxin reduct