Zobrazeno 1 - 2
of 2
pro vyhledávání: '"Andrew L.H. Webster"'
Autor:
Sunandini Sridhar, David I. Kutler, Bhuvanesh Singh, Susanne I. Wells, Ji-Dung Luo, Mathijs A. Sanders, Margaret L. MacMillan, Ashlyn-Maree Gonzalez, Lorenzo Bean, Rebecca Tryon, Huasong Tian, Jordi Surrallés, Arleen D. Auerbach, Kevin Hadi, Moonjung Jung, Ralf Dietrich, Matthew M. Edwards, Eunike Velleuer, Krupa R. Patel, Frank X. Donovan, Amnon Koren, Marcin Imielinski, Audrey Goldfarb, Ozgur Rosti, Jeffrey C. Rastatter, Theresa Scognamiglio, John E. Wagner, Andrew L.H. Webster, Maria Cancio, Olivier Fedrigo, Agata Smogorzewska, Jennifer A. Kennedy, Thomas Carrol, Grover C. Bagby, Joel Rosiene, Allana Rosenberg, Thomas Heineman, Ryan R. White, Raymond J. Noonan, Farid Boulad, Francis P. Lach, Settara C. Chandrasekharappa, Peter J. Campbell, Roger D. Vaughan
Fanconi anemia (FA), a model syndrome of genome instability, is caused by a deficiency in DNA interstrand crosslink (ICL) repair resulting in chromosome breakage1–3. The FA repair pathway comprises at least 22 FANC proteins including BRCA1 and BRCA
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::d9afd781051fca6c3cb7bdbe0af4e0c0
https://doi.org/10.1101/2021.08.14.456365
https://doi.org/10.1101/2021.08.14.456365
Publikováno v:
Canadian Journal of Cardiology. 29:46-57
A commonly-assumed paradigm holds that the primary genetic determinant of cardiovascular disease resides within the DNA sequence of our genes. This paradigm can be challenged. For example, how do sequence changes in the non-coding region of the genom
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::28742a8b3e1e5dc896280d39a9e5f840
https://doi.org/10.1016/j.cjca.2012.10.023
https://doi.org/10.1016/j.cjca.2012.10.023
