Zobrazeno 1 - 10
of 13
pro vyhledávání: '"Andrew H. Lundberg"'
Autor:
Kazuhiko Fukatsu, Malak Kotb, Omaima M. Sabek, Lillian W. Gaber, Christopher S. Callicutt, A. Osama Gaber, Henry G. Wilcox, Andrew H. Lundberg
Publikováno v:
Surgery. 133:186-196
Lung injury in severe acute pancreatitis is mediated by infiltrating leukocytes. Our laboratory has previously demonstrated that acute lung injury in acute pancreatitis results in an up-regulation of vascular adhesion molecule-1 (VCAM-1) cell surface
Autor:
Cheryl D. Johnson, Ben L. Zarzaur, Kazuhiko Fukatsu, Henry G. Wilcox, Kenneth A. Kudsk, Andrew H. Lundberg
Publikováno v:
Annals of Surgery. 233:660-668
Objective To determine whether parenteral feeding (IV-TPN) influences the local and systemic response to an intestinal insult. Background Data Parenteral feeding increases ICAM-1 expression and attracts neutrophils (PMNs) to the intestine compared wi
Autor:
Cheryl D. Johnson, Andrew H. Lundberg, Ben L. Zarzaur, Kazuhiko Fukatsu, Kenneth A. Kudsk, M. Keith Hanna, Henry G. Wilcox
Publikováno v:
Journal of Surgical Research. 97:41-48
Background. Total parenteral nutrition (IV-TPN) increases neutrophil accumulation in the small intestine, expression of intestinal ICAM-1 and P-selectin, and upregulates E-selectin expression in the lung. Endothelial activation induced by lack of ent
Autor:
A. Osama Gaber, Malak Kotb, Lillian W. Gaber, Andrew H. Lundberg, Scott Callicutt, Kazuhiko Fukatsu, Kenneth A. Kudsk, Henry G. Wilcox
Publikováno v:
Annals of Surgery. 233:213-220
The high rates of death and complications associated with severe acute pancreatitis (AP) stem from an overwhelming inflammatory response that can cause injury to distant organs such as lung tissue. 1,2 Patients with severe AP develop pulmonary dysfun
Autor:
Andrew H. Lundberg, A O Gaber, Lillian W. Gaber, Malak Kotb, Omaima M. Sabek, James Henry, Anna Norby-Teglund, James W. Eubanks
Publikováno v:
Pancreas. 21:41-51
Acute pancreatitis (AP) is characterized by release of proteolytic enzymes from the pancreas and a powerful inflammatory cytokine cascade that mediates the systemic manifestations and contributes to the mortality of the disease. The purpose of this s
Autor:
A O Gaber, Omaima M. Sabek, Malak Kotb, James A. Russell, Lillian W. Gaber, S Callicutt, N Granger, Andrew H. Lundberg
Publikováno v:
Journal of Gastrointestinal Surgery. 4:248-257
Lung injury is a major cause of patient morbidity in acute pancreatitis. The purpose of this study was to examine the mechanism of pulmonary infiltration and lung injury in acute pancreatitis. Mice were fed a choline-deficient/ethionine-supplemented
Autor:
D. Neil Granger, Lillian W. Gaber, Janice Russell, A. Osama Gaber, Malak Kotb, Andrew H. Lundberg, Omaima M. Sabek, James Henry
Publikováno v:
Annals of Surgery. 231:213-222
Acute pancreatitis (AP) is a multiple system disorder that, in its severe state, has manifestations sometimes indistinguishable from those seen in septic shock. Like sepsis, the death and complications related to this disease are due to the respirato
Autor:
Cheryl D. Johnson, Kazuhiko Fukatsu, Ben L. Zarzaur, Andrew H. Lundberg, Keith M. Hanna, Kenneth A. Kudsk, Yong Wu, Henry G. Wilcox
Publikováno v:
Shock (Augusta, Ga.). 15(1)
The gut primes neutrophils (PMNs) during injury, which can then induce distant organ damage after a second insult. ICAM-1 is an important adhesion molecule in PMN attachment to the vascular endothelium. Parenteral nutrition (TPN) decreases gut levels
Autor:
Henry G. Wilcox, Kazuhiko Fukatsu, Kenneth A. Kudsk, D. N. Granger, M. K. Hanna, Yong Wu, A. O. Gaber, Andrew H. Lundberg
Publikováno v:
Archives of surgery (Chicago, Ill. : 1960). 135(10)
Hypothesis Intravenous total parenteral nutrition (TPN) induces intestinal polymorphonuclear neutrophil recruitment with increased intestinal intercellular adhesion molecule-1 expression. While intercellular adhesion molecule-1 causes firm adhesion o
Autor:
Yong Wu, M. Keith Hanna, Kazuhiko Fukatsu, Andrew H. Lundberg, Kenneth A. Kudsk, Henry G. Wilcox, A. Osama Gaber, D. Neil Granger
Publikováno v:
Archives of surgery (Chicago, Ill. : 1960). 134(10)
Hypothesis The levels of intestinal interleukin 10 and interleukin 4, inhibitors of intercellular adhesion molecule-1 (ICAM-1) expression, decline with total parenteral nutrition (TPN). These cytokine changes induced by lack of enteral nutrition may