Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Andrew Caton"'
Autor:
Felipe Bedoya, Guang-Shing Cheng, Abigail Leibow, Nardine Zakhary, Katherine Weissler, Victoria Garcia, Elizabeth Kropf, Malinda Aitken, David Garlick, E. Wherry, Jan Erikson, Andrew Caton
Publikováno v:
The Journal of Immunology. 190:139.11-139.11
We have examined the formation, participation and functional specialization of virus-reactive Foxp3+ regulatory T cells (Tregs) in a mouse model of influenza virus infection. “Natural” Tregs generated intra-thymically based on interactions with a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::17934ba5e3043ec87269ddd55fde7db8
https://doi.org/10.4049/jimmunol.190.supp.139.11
https://doi.org/10.4049/jimmunol.190.supp.139.11
Publikováno v:
The Journal of Immunology. 188:65.2-65.2
It is generally accepted that developing thymocytes can become Foxp3+ regulatory T cells (Tregs) through recognition of self-antigens during thymic selection. Tregs can also develop from conventional CD4+ T cells responding to exogenously administere
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::e8478ecc4b2b9370a019881e36ecdc89
https://doi.org/10.4049/jimmunol.188.supp.65.2
https://doi.org/10.4049/jimmunol.188.supp.65.2
Publikováno v:
The Journal of Immunology. 188:60.8-60.8
Th17 cells have been implicated in a number of autoimmune and inflammatory disorders, yet signals that can lead to a Th17 response in vivo are poorly understood. We have analyzed this question using a transgenic mouse model in which autoreactive CD4+
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::d46d2bf225daed792bb280bfb9583ee3
https://doi.org/10.4049/jimmunol.188.supp.60.8
https://doi.org/10.4049/jimmunol.188.supp.60.8
Publikováno v:
The Journal of Immunology. 188:60.6-60.6
CD4+ T cells make a crucial contribution to the development of inflammatory arthritis both in humans and in mouse models. However, how the affinity with which T cells recognize target antigens might shape disease development and influence treatment m
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::a8d7f104a2756e1b5d65d65efac55409
https://doi.org/10.4049/jimmunol.188.supp.60.6
https://doi.org/10.4049/jimmunol.188.supp.60.6
Publikováno v:
The Journal of Immunology. 186:101.23-101.23
Th17 cells have been implicated in a number of autoimmune diseases and their animal models including inflammatory arthritis. How these cells arise in the context of autoimmunity is not well understood. We recently described a mouse model of autoimmun
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::140bdd6d5b797ae8f9085bf5bae24aba
https://doi.org/10.4049/jimmunol.186.supp.101.23
https://doi.org/10.4049/jimmunol.186.supp.101.23
Autor:
Kathryn Juchem, Britt Anderson, Cuiling Zhang, Jennifer McNiff, Anthony Demetris, Andrew Caton, Warren Shlomchik, Mark Shlomchik
Publikováno v:
The Journal of Immunology. 186:169.6-169.6
Graft versus host disease (GVHD) is a major complication of allogeneic stem cell transplantation (alloSCT). In murine models of alloSCT, naïve T cells (TN) cause GVHD, while effector memory T cells (TEM) do not. Why TEM fail to cause GVHD is unknow
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::9e27c770f4cd15592eb095df6a1d4134
https://doi.org/10.4049/jimmunol.186.supp.169.6
https://doi.org/10.4049/jimmunol.186.supp.169.6