Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Andrea E. Vasconez"'
Autor:
Christian Fork, Andrea E. Vasconez, Patrick Janetzko, Carlo Angioni, Yannick Schreiber, Nerea Ferreirós, Gerd Geisslinger, Matthias S. Leisegang, Dieter Steinhilber, Ralf P. Brandes
Publikováno v:
Journal of Lipid Research, Vol 58, Iss 2, Pp 386-392 (2017)
Nonsteroidal anti-inflammatory drugs are the most widely used medicine to treat pain and inflammation, and to inhibit platelet function. Understanding the expression regulation of enzymes of the prostanoid pathway is of great medical relevance. Histo
Externí odkaz:
https://doaj.org/article/9bf37fdf2b6e410a8cb48d901a0dbe37
Autor:
Ralf P. Brandes, Andrea E. Vasconez, Stefan Offermanns, Katalin Pálfi, Eunjee Lee, Andreas Weigert, Jun Zhu, Christoph Schürmann, Praveen Mathoor, Matthias S. Leisegang, Christoph Kruse, Bart van de Sluis, James A. Oo, Shengpeng Wang, Giulia K. Buchmann, Franziska Luise Dienst, Patrick O. Humbert, Stefan Günther, Ilka Wittig
Publikováno v:
Cardiovascular Research, 115(14), 1963-1974. Oxford University Press
Aims The protein Scrib (Scribble 1) is known to control apico-basal polarity in epithelial cells. The role of polarity proteins in the vascular system remains poorly characterized; however, we previously reported that Scrib maintains the endothelial
Autor:
Klaus Badenhoop, Christoph Schürmann, Juergen Konczalla, Sepide Kashefiolasl, Geert Carmeliet, Matthias S. Leisegang, Ralf P. Brandes, Volker Seifert, Beatrice Pflüger-Müller, Gudrun Hintereder, Katrin Schröder, Andrea E. Vasconez, Valeska Helfinger, Voahanginirina Randriamboavonjy
Publikováno v:
Neurosurgery
BACKGROUND: Cerebral vasospasm (CVS) is a frequent complication after subarachnoid hemorrhage (SAH), with no sufficient therapy and a complex pathophysiology. OBJECTIVE: To explore the vitamin D system as a potential treatment for CVS. METHODS: 25-vi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7a6b2e63938ce6f8de9bd3a3f566d5f3
https://lirias.kuleuven.be/handle/123456789/665109
https://lirias.kuleuven.be/handle/123456789/665109
Autor:
Ralf P. Brandes, Carlo Angioni, Patrick Janetzko, Yannick Schreiber, Dieter Steinhilber, Andrea E. Vasconez, Christian Fork, Gerd Geisslinger, Nerea Ferreirós, Matthias S. Leisegang
Publikováno v:
Journal of Lipid Research, Vol 58, Iss 2, Pp 386-392 (2017)
Nonsteroidal anti-inflammatory drugs are the most widely used medicine to treat pain and inflammation, and to inhibit platelet function. Understanding the expression regulation of enzymes of the prostanoid pathway is of great medical relevance. Histo
Autor:
Elisabetta Gamen, Mario Looso, Ralf P. Brandes, Andrea E. Vasconez, Christian Fork, James A. Oo, Sandra Seredinski, Juliane Hitzel, Matthias S. Leisegang, Wei Chen, Ivana Josipovic, Beatrice Pflüger, Dagmar Meyer zu Heringdorf, Soni Savai Pullamsetti
Publikováno v:
Journal of molecular and cellular cardiology. 116
Sphingosine-1-Phosphate (S1P) is a potent signaling lipid. The effects of S1P are mediated by the five S1P receptors (S1PR). In the endothelium S1PR1 is the predominant receptor and thus S1PR1 abundance limits S1P signaling. Recently, lncRNAs were id
Autor:
Jens Preussner, Mario Looso, Christian Fork, Gerd Geisslinger, Dijana Iloska, Nerea Ferreirós, Dominique Thomas, Andrea E. Vasconez, Kim-Kristin Prior, Ralf P. Brandes, Soni Savai Pullamsetti, Matthias S. Leisegang, Sandra Labocha, Carlo Angioni, Dieter Steinhilber, Ivana Josipovic
Publikováno v:
Acta physiologica (Oxford, England). 218(1)
AIM Platelet-activating factor acetyl hydrolase 1B1 (PAFAH1B1, also known as Lis1) is a protein essentially involved in neurogenesis and mostly studied in the nervous system. As we observed a significant expression of PAFAH1B1 in the vascular system,
Autor:
Katrin Schröder, Gerd Geisslinger, Ralf P. Brandes, Kristian Helin, Christian Fork, Corina Ratiu, Ingrid Fleming, Andrea E. Vasconez, Patrick Janetzko, Matthias S. Leisegang, Lunda Gu, James A. Oo, Beatrice Pflüger-Müller
Publikováno v:
Acta Physiologica. 225:e13168
AIM The histone demethylase Jarid1b limits gene expression by removing the active methyl mark from histone3 lysine4 at gene promoter regions. A vascular function of Jarid1b is unknown, but a vasoprotective function to inflammatory and hypertrophic st