Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Andrea E. M. Stinghen"'
Autor:
Elberth M. Schiefer, Andressa F. Santos, Marcia Muller, Andrea E. M. Stinghen, Lucas H. Negri, Jose L. Fabris
Publikováno v:
2022 SBFoton International Optics and Photonics Conference (SBFoton IOPC).
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::2233a3824d4a5ec15e29029639a8d982
https://doi.org/10.1109/sbfotoniopc54450.2022.9992542
https://doi.org/10.1109/sbfotoniopc54450.2022.9992542
Autor:
Paulo C. Gregório, Gilson Biagini, Regiane S. da Cunha, Júlia Budag, Ana Maria Martins, Lara Valiño Rivas, Elberth M. Schiefer, Maria Dolores Sánchez-Niño, Alberto Ortiz, Andréa E. M. Stinghen, Fellype C. Barreto
Publikováno v:
Kidney & Blood Pressure Research, Pp 1-10 (2022)
Background and Aims: Inflammation and endothelial damage play a pivotal role in Fabry disease (FD) manifestations. In daily clinical practice, FD is mainly monitored by traditional biomarkers of target organ injury, such as serum creatinine and prote
Externí odkaz:
https://doaj.org/article/8ff34d0a10fe41c88d672644d3fe72b9
Publikováno v:
Brazilian Journal of Nephrology, Iss 0 (2018)
Externí odkaz:
https://doaj.org/article/ead0503526b34d10a22434ceade2ac17
Autor:
Lisienny C. T. Rempel, Alessandra B. Finco, Rayana A. P. Maciel, Bruna Bosquetti, Larissa M. Alvarenga, Wesley M. Souza, Roberto Pecoits-Filho, Andréa E. M. Stinghen
Publikováno v:
Toxins, Vol 7, Iss 5, Pp 1722-1737 (2015)
Advanced glycation end products (AGEs) are compounds classified as uremic toxins in patients with chronic kidney disease that have several pro-inflammatory effects and are implicated in the development of cardiovascular diseases. To explore the mecha
Externí odkaz:
https://doaj.org/article/230e92c9b720470fbee4cb55b9b3b656
Publikováno v:
Toxins, Vol 6, Iss 6, Pp 1742-1760 (2014)
Endothelial dysfunction plays a key role in stroke in chronic kidney disease patients. To explore the underlying mechanisms, we evaluated the effects of two uremic toxins on cerebral endothelium function. bEnd.3 cells were exposed to indoxyl sulfate
Externí odkaz:
https://doaj.org/article/2775be8707a04565946c3b9434fa0cd7
Autor:
Rayana A. P. Maciel, Regiane S. Cunha, Valentina Busato, Célia R. C. Franco, Paulo C. Gregório, Carla J. R. Dolenga, Lia S. Nakao, Ziad A. Massy, Agnès Boullier, Roberto Pecoits-Filho, Andréa E. M. Stinghen
Publikováno v:
Toxins, Vol 10, Iss 10, p 404 (2018)
Endothelial dysfunction in uremia can result in cell-to-cell junction loss and increased permeability, contributing to cardiovascular diseases (CVD) development. This study evaluated the impact of the uremic milieu on endothelial morphology and cell
Externí odkaz:
https://doaj.org/article/497580018be6486e8da4ebb14837f243
Autor:
Ligia Maria Claro, Andrea N. Moreno-Amaral, Ana Carolina Gadotti, Carla J. Dolenga, Lia S. Nakao, Marina L.V. Azevedo, Lucia de Noronha, Marcia Olandoski, Thyago P. de Moraes, Andréa E. M. Stinghen, Roberto Pécoits-Filho
Publikováno v:
Toxins, Vol 10, Iss 10, p 384 (2018)
Uremic toxin (UT) retention in chronic kidney disease (CKD) affects biological systems. We aimed to identify the associations between UT, inflammatory biomarkers and biomarkers of the uremic cardiovascular response (BUCVR) and their impact on cardiov
Externí odkaz:
https://doaj.org/article/866804be73dc4ec7a5de8f67fe3593f1
Publikováno v:
International Journal of Hypertension, Vol 2011 (2011)
Chronic kidney disease (CKD) is highly prevalent and a multiplier of cardiovascular disease (CVD) and cannot be completely explained by traditional Framinghan risk factors. Consequently, greater emphasis has been placed in nontraditional risk factors
Externí odkaz:
https://doaj.org/article/f6e421510dfd435daf99ea0d96e503a5