Zobrazeno 1 - 10
of 17
pro vyhledávání: '"Andrea Anedda"'
Autor:
M Mar González-Barroso, Irina Giurgea, Fredéric Bouillaud, Andrea Anedda, Christine Bellanné-Chantelot, Laurence Hubert, Yves de Keyzer, Pascale de Lonlay, Daniel Ricquier
Publikováno v:
PLoS ONE, Vol 3, Iss 12, p e3850 (2008)
Although the most common mechanism underlying congenital hyperinsulinism is dysfunction of the pancreatic ATP-sensitive potassium channel, the pathogenesis and genetic origins of this disease remains largely unexplained in more than half of all patie
Externí odkaz:
https://doaj.org/article/1d46ac5c1e6e4f8aa527d7b2e5f7cdd6
Publikováno v:
Biochimica et Biophysica Acta (BBA) - Bioenergetics. 1859:e11-e12
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::ff11482cc303fea9a36c7ae29fc01bc5
https://doi.org/10.1016/j.bbabio.2018.09.036
https://doi.org/10.1016/j.bbabio.2018.09.036
Autor:
Andrea Anedda, Susana Cadenas, Bárbara Acosta-Iborra, Elia López-Bernardo, M.Saadeh Suleiman, Manuel O. Landázuri
Publikováno v:
Free Radical Biology and Medicine. 61:395-407
Uncoupling protein 3 (UCP3) is a member of the mitochondrial inner membrane carrier superfamily that modulates energy efficiency by catalyzing proton conductance and thus decreasing the production of superoxide anion. However, its role during oxidati
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ef2af599bc4a7f6ca784cb5148bb7847
https://doi.org/10.1016/j.freeradbiomed.2013.04.007
https://doi.org/10.1016/j.freeradbiomed.2013.04.007
Autor:
Eunate Gallardo-Vara, Eduardo Rial, Mariano Redondo-Horcajo, Andrea Anedda, Leonor Rodríguez-Sánchez, M. Mar González-Barroso
Publikováno v:
Digital.CSIC. Repositorio Institucional del CSIC
instname
instname
8 páginas, 4 figuras -- PAGS nros. 1768-1775
While metformin has been widely used to treat type 2 diabetes for the last fifty years, its mode of action remains unclear. Hence, we investigated the short-term alterations in energy metabolism caus
While metformin has been widely used to treat type 2 diabetes for the last fifty years, its mode of action remains unclear. Hence, we investigated the short-term alterations in energy metabolism caus
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fc7f1684f0a25fc1b666efac3e9fa195
https://doi.org/10.1016/j.bbabio.2012.02.019
https://doi.org/10.1016/j.bbabio.2012.02.019
Publikováno v:
Free Radical Biology and Medicine. 120:S32
Mitochondrial uncoupling proteins (UCPs) are able to increase the mitochondrial inner membrane proton conductance and consequently decrease the electrochemical proton gradient, thus diminishing the efficiency of oxidative phosphorylation. UCP3 is exp
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::78cd043031a1b9784e605252e41a2c46
https://doi.org/10.1016/j.freeradbiomed.2018.04.112
https://doi.org/10.1016/j.freeradbiomed.2018.04.112
Autor:
Susana Cadenas, Andrea Anedda, Patricia Sánchez-Pérez, Elia López-Bernardo, Bárbara Acosta-Iborra
Publikováno v:
Free radical biologymedicine. 88
4-Hydroxy-2-nonenal (HNE) is a highly cytotoxic product of lipid peroxidation. Nevertheless, at low concentrations, it is able to mediate cell signaling and to activate protective pathways, including that of the transcription factor Nrf2 (nuclear fac
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e052f476f41f035972b47aae73a4f2a9
https://pubmed.ncbi.nlm.nih.gov/25843654
https://pubmed.ncbi.nlm.nih.gov/25843654
Autor:
Patricia Sánchez-Pérez, Bárbara Acosta-Iborra, Susana Cadenas, Elia López-Bernardo, Andrea Anedda
Publikováno v:
Biochimica et Biophysica Acta (BBA) - Bioenergetics. 1857:e79
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::e767a1f7901cb352fe8945c1fad48782
https://doi.org/10.1016/j.bbabio.2016.04.326
https://doi.org/10.1016/j.bbabio.2016.04.326
Publikováno v:
Biochimica et Biophysica Acta (BBA) - Bioenergetics. 1797
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0145cfc1e186311707c0e87694b2a5b8
Autor:
Yves de Keyzer, Pascale de Lonlay, Christine Bellanné-Chantelot, Andrea Anedda, Daniel Ricquier, Irina Giurgea, Laurence Hubert, M. Mar González-Barroso, Frédéric Bouillaud
Publikováno v:
PLoS ONE
PLoS ONE, Vol 3, Iss 12, p e3850 (2008)
Digital.CSIC. Repositorio Institucional del CSIC
instname
PLoS ONE, Vol 3, Iss 12, p e3850 (2008)
Digital.CSIC. Repositorio Institucional del CSIC
instname
5 figuras, 1 tabla
Although the most common mechanism underlying congenital hyperinsulinism is dysfunction of the pancreatic ATP-sensitive potassium channel, the pathogenesis and genetic origins of this disease remains largely unexplained in mor
Although the most common mechanism underlying congenital hyperinsulinism is dysfunction of the pancreatic ATP-sensitive potassium channel, the pathogenesis and genetic origins of this disease remains largely unexplained in mor
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::dbe4737a1d88c2e77abcf8347b391d99
https://pubmed.ncbi.nlm.nih.gov/19065272
https://pubmed.ncbi.nlm.nih.gov/19065272
Publikováno v:
Digital.CSIC. Repositorio Institucional del CSIC
instname
instname
8 páginas, 4 figuras -- PAGS nros. 33-40
Metformin is a drug widely used to treat type 2 diabetes. It enhances insulin sensitivity by improving glucose utilization in tissues like liver or muscle. Metformin inhibits respiration, and the decreas
Metformin is a drug widely used to treat type 2 diabetes. It enhances insulin sensitivity by improving glucose utilization in tissues like liver or muscle. Metformin inhibits respiration, and the decreas
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e22dac6ea5d2757fbe9e4fc395e0c46b
http://hdl.handle.net/10261/66283
http://hdl.handle.net/10261/66283
