Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Anatoly Prokvolit"'
Autor:
Michelle Haber, Andrei V. Gudkov, Murray D. Norris, Glenn M. Marshall, Alan C. Sartorelli, Katerina V. Gurova, Pavel G. Komarov, Nadezhda Isachenko, Andrei Purmal, Janice Smith, Claudia Flemming, Chengyuan Xue, Anatoly Prokvolit, Marina Pajic, Jayne Murray, Fujiko Watt, Catherine A. Burkhart
Supplementary Table 1, Supplementary Figures 1-5 from Small-Molecule Multidrug Resistance–Associated Protein 1 Inhibitor Reversan Increases the Therapeutic Index of Chemotherapy in Mouse Models of Neuroblastoma
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ac22996ef79fe9d1361b986939ba03a8
https://doi.org/10.1158/0008-5472.22378460.v1
https://doi.org/10.1158/0008-5472.22378460.v1
Autor:
Janice Smith, Michelle Haber, Nadezhda Isachenko, Andrei Purmal, Anatoly Prokvolit, Catherine Burkhart, Marina Pajic, Chengyuan Xue, Murray D. Norris, Pavel G. Komarov, Andrei V. Gudkov, Claudia Flemming, Katerina Gurova, Jayne Murray, Alan C. Sartorelli, Glenn M. Marshall, Fujiko Watt
Publikováno v:
Cancer Research. 69:6573-6580
The multidrug resistance–associated protein 1 (MRP1) has been closely linked to poor treatment response in several cancers, most notably neuroblastoma. Homozygous deletion of the MRP1 gene in primary murine neuroblastoma tumors resulted in increase
Autor:
Petro Ryabokon, Anatoly Prokvolit, Natalya V. Narizhneva, Katerina Gurova, Pavel G. Komarov, Andrei V. Gudkov, Andrei Purmal, Inna Shyshynova, Natalia D. Tararova
Publikováno v:
Cell cycle (Georgetown, Tex.). 8(24)
In prostate cancer (PCa) patients, initial responsiveness to androgen deprivation therapy is frequently followed by relapse due to development of treatment-resistant androgen-independent PCa. This is typically associated with acquisition of mutations
Autor:
Tom R. Webb, Anatoly Prokvolit, Eugenia Samoylova, Andrei V. Gudkov, Natalia D. Tararova, Dmitriy Lvovskiy, Mahrukh K. Ganapathi, Canhui Guo, Dmitry A. Bosykh, Lyudmila G. Burdelya, Ram Ganapathi, Katerina Gurova, Anna V. Khodyakova, George R. Stark, Jason E. Hill
Renal cell carcinomas (RCC) commonly retain wild-type but functionally inactive p53, which is repressed by an unknown dominant mechanism. To help reveal this mechanism, we screened a diverse chemical library for small molecules capable of restoring p
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3a0b1ccbf5e4b3ed6dfcbdfa07da50ef
https://europepmc.org/articles/PMC1297696/
https://europepmc.org/articles/PMC1297696/
Publikováno v:
Melanoma Research. 16:S80