Zobrazeno 1 - 10
of 39
pro vyhledávání: '"Anabel Heiniger"'
Autor:
Jose Roman-Gomez, Antonio Jimenez-Velasco, Xabier Agirre, Juan A. Castillejo, German Navarro, Edurne San Jose-Eneriz, Leire Garate, Lucia Cordeu, Francisco Cervantes, Felipe Prosper, Anabel Heiniger, Antonio Torres
Publikováno v:
Haematologica, Vol 92, Iss 2 (2007)
Background and Objectives Cancer testis antigens (CTA) provide attractive targets for cancer-specific immunotherapy. Although CTA genes are expressed in some normal tissues, such as the testis, this immunologically protected site lacks MHC I expressi
Externí odkaz:
https://doaj.org/article/85e85d67708243f6ab0179ad85b37842
Autor:
Felipe Prósper, José Román-Gómez, Puri Fortes, María José Calasanz, Anabel Heiniger, Antonio Torres, Jesús García-Foncillas, Ignacio Pérez-Roger, Amaia Vilas-Zornoza, Germán Navarro, Borja Saez, Oscar Aparicio, Lucia Cordeu, Eva Bandrés, Leire Garate, Edurne San José-Enériz, Antonio Jiménez-Velasco, Xabier Agirre
Supplementary Table S3 from Down-Regulation of hsa-miR-10a in Chronic Myeloid Leukemia CD34+ Cells Increases USF2-Mediated Cell Growth
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7d9d3b004a16b81b5ff09bea9c13d327
https://doi.org/10.1158/1541-7786.22517101
https://doi.org/10.1158/1541-7786.22517101
Autor:
Felipe Prósper, José Román-Gómez, Puri Fortes, María José Calasanz, Anabel Heiniger, Antonio Torres, Jesús García-Foncillas, Ignacio Pérez-Roger, Amaia Vilas-Zornoza, Germán Navarro, Borja Saez, Oscar Aparicio, Lucia Cordeu, Eva Bandrés, Leire Garate, Edurne San José-Enériz, Antonio Jiménez-Velasco, Xabier Agirre
Supplementary Figures S1-S6 from Down-Regulation of hsa-miR-10a in Chronic Myeloid Leukemia CD34+ Cells Increases USF2-Mediated Cell Growth
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::970aa5b884fe8ab2c3cb3a517fe10f8b
https://doi.org/10.1158/1541-7786.22517110
https://doi.org/10.1158/1541-7786.22517110
Autor:
Felipe Prósper, José Román-Gomez, Reiner Siebert, Antonio Torres, Anabel Heiniger, Vanesa Martín, María José Calasanz, Eva Bandrés, José Rifón, Puri Fortes, Paula Rodríguez-Otero, Gloria Abizanda, Edurne San José-Eneriz, Leire Gárate, Lucia Cordeu, José Ignacio Martin-Subero, Antonio Jiménez-Velasco, Amaia Vilas-Zornoza, Xabier Agirre
Supplementary Methods, Figures 1-3, Tables 1-6 from Epigenetic Silencing of the Tumor Suppressor MicroRNA Hsa-miR-124a Regulates CDK6 Expression and Confers a Poor Prognosis in Acute Lymphoblastic Leukemia
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0b7c37daa27675e6678d1b186c6c0a30
https://doi.org/10.1158/0008-5472.22377672
https://doi.org/10.1158/0008-5472.22377672
Autor:
C. E. López-Jorge, G. Sánchez, Manuel Barrios, S. de la Iglesia, María Teresa Gómez-Casares, Anabel Heiniger, T. Ramírez, Jacobo Patiño López, T. Molero, Miguel A. García-Bello, Antonio Jiménez-Velasco
Publikováno v:
Annals of Hematology. 91:1245-1250
The level of BCR-ABL1 reached after treatment with tyrosine kinase inhibitors is an effective marker of the therapeutic response and a good survival predictor in chronic myeloid leukemia (CML) patients. However, no agreement has yet been achieved abo
Autor:
Felipe Prosper, Francisco Cervantes, Jose A. Martinez-Climent, Vicente Fresquet, Antonio Torres, Xabier Agirre, Victor Arqueros, Jose Roman-Gomez, Edurne San José-Enériz, Vanesa Martin, Leire Garate, Antonio Jiménez-Velasco, Lucia Cordeu, Anabel Heiniger
Publikováno v:
European Journal of Cancer. 45:1877-1889
Background Expression of the pro-apoptotic BCL-2-interacting mediator ( BIM ) has recently been implicated in imatinib-induced apoptosis of BCR-ABL1 + cells. However, the mechanisms involved in the regulation of BIM in CML and its role in the clinica
Autor:
Xabier Agirre, Oscar Aparicio, Jesús García-Foncillas, German Navarro, Amaia Vilas-Zornoza, Ignacio Pérez-Roger, Antoni Torres, Antonio Jiménez-Velasco, María José Calasanz, Lucia Cordeu, Felipe Prosper, Puri Fortes, Eva Bandrés, Edurne San José-Enériz, Leire Garate, Jose Roman-Gomez, Anabel Heiniger, Borja Saez
Publikováno v:
Molecular Cancer Research. 6:1830-1840
MicroRNAs (miRNA) are small noncoding, single-stranded RNAs that inhibit gene expression at a posttranscriptional level, whose abnormal expression has been described in different tumors. The aim of our study was to identify miRNAs potentially implica
Autor:
Anabel Heiniger, Edurne San José-Enériz, Lucia Cordeu, Jose Roman-Gomez, Vanesa Martin, Xabier Agirre, Amaia Vilas-Zornoza, Leire Garate, Juan A. Castillejo, Antoni Torres, Antonio Jiménez-Velasco, Felipe Prosper
Publikováno v:
Dadun. Depósito Académico Digital de la Universidad de Navarra
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The clinical significance of aberrant promoter methylation of the canonical Wnt pathway antagonist genes (sFRP1, sFRP2, sFRP4, sFRP5, Wif1, Dkk3, and Hdpr1) and also putative tumor-suppressor gene Wnt5a, belonging to the non-canonical Wnt signaling p
Autor:
Xabier Agirre, Isabel M. Isidro, Antoni Torres, Jose Roman-Gomez, Lucia Cordeu, Enrique J. Andreu, Antonio Jiménez-Velasco, Angel Rubio, Leire Garate, Manel Esteller, Esteban Ballestar, Anabel Heiniger, Elizabeth Guruceaga, Norma C. Gutiérrez, Ignacio Pérez-Roger, María José Calasanz, Edurne San José-Enériz, Felipe Prosper
Publikováno v:
British Journal of Haematology. 142:571-582
In order to determine new signal transduction pathways implicated in chronic myeloid leukaemia (CML), we performed a gene expression profile comparison between CD34+ cells from CML patients and healthy donors. Functional studies were performed using
Autor:
Jose Roman-Gomez, Edurne San José-Enériz, Cristina Montiel-Duarte, Antoni Torres, María José Calasanz, Xabier Agirre, Felipe Prosper, Leire Garate, Anabel Heiniger, Lucia Cordeu, Enrique J. Andreu, Antonio Jiménez-Velasco
Publikováno v:
Leukemia Research. 32:709-716
The aim of our study was to determine the potential mechanism(s) implicated in Imatinib resistance in patients with Ph + ALL. Resistance of Ph + ALL cells to Imatinib-induced apoptosis was associated with lack of inhibition of Akt phosphorylation. Ad