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Autor:
Yael Borovitz, Daniel Landau, Amit Dagan, Hadas Alfandari, Orly Haskin, Shelly Levi, Gilad Hamdani, Daniella Levy Erez, Shimrit Tzvi-Behr, Jenny Weinbrand-Goichberg, Ana Tobar Foigelman, Ruth Rahamimov
Publikováno v:
Frontiers in Pediatrics, Vol 12 (2024)
BackgroundC3 Glomerulopathy (C3G) is a complement-mediated disease, with predominant C3 deposits, where pathogenic genetic variants in complement system components and circulating autoantibodies result in loss of control of the alternative pathway, h
Externí odkaz:
https://doaj.org/article/3d9a585fb5004c1482979bea2b126c24