Zobrazeno 1 - 10
of 14
pro vyhledávání: '"Ana Paula Ferragut Cardoso"'
Autor:
Puttappa R. Dodmane, Lora L. Arnold, Karen L. Pennington, Rakesh K. Singh, Ana Paula Ferragut Cardoso, Samuel M. Cohen
Publikováno v:
Toxicology Reports, Vol 2, Iss C, Pp 833-837 (2015)
Chronic exposure to high levels of inorganic arsenic (iAs) has been associated with cancerous and non-cancerous health effects, including cardiovascular effects. However, the mechanism for a presumed toxic effect of arsenic on vascular tissue is not
Externí odkaz:
https://doaj.org/article/3e3e86732cee4d3b8e0f65c7f1bd293e
Autor:
Mayukh Banerjee, Angeliki Lykoudi, Alexandra N Nail, J. Christopher States, Ana Paula Ferragut Cardoso
Publikováno v:
Semin Cancer Biol
Genomic instability consists of a range of genetic alterations within the genome that contributes to tumor heterogeneity and drug resistance. It is a well-established characteristic of most cancer cells. Genome instability induction results from defe
Autor:
J. Christopher States, Jianmin Pan, Mayukh Banerjee, Laila Al-Eryani, Sudhir Srivastava, Theodore S. Kalbfleisch, Shesh N. Rai, Ana Paula Ferragut Cardoso
Publikováno v:
Archives of Toxicology
Chronic arsenic exposure causes skin cancer, although the underlying molecular mechanisms are not well defined. Altered microRNA and mRNA expression likely play a pivotal role in carcinogenesis. Changes in genome-wide differential expression of miRNA
Autor:
Samuel M. Cohen, Lígia M M Gomide, Merielen Garcia Nascimento e Pontes, Nathália Pereira de Souza, João Lauro Viana de Camargo, Carlos Márcio Nóbrega de Jesus, Ana Paula Ferragut Cardoso, Lora L. Arnold
Publikováno v:
Int J Exp Pathol
Scopus
Repositório Institucional da UNESP
Universidade Estadual Paulista (UNESP)
instacron:UNESP
Scopus
Repositório Institucional da UNESP
Universidade Estadual Paulista (UNESP)
instacron:UNESP
Made available in DSpace on 2021-06-25T10:50:05Z (GMT). No. of bitstreams: 0 Previous issue date: 2021-02-01 Cryptorchidism is one of the main risk factors for infertility and testicular cancer. Orchiopexy surgery corrects cryptorchidism effects. Dif
Autor:
Daniel W. Wilkey, J. Christopher States, Michael L. Merchant, Ageliki Lykoudi, Walter H. Watson, Jianmin Pan, Mayukh Banerjee, Shesh N. Rai, Nichola C. Garbett, Ana Paula Ferragut Cardoso
Publikováno v:
Chem Res Toxicol
Exposure to arsenic, a class I carcinogen, affects 200 million people globally. Skin is the major target organ, but the molecular etiology of arsenic-induced skin carcinogenesis remains unclear. Arsenite (As3+)-induced disruption of alternative splic
Publikováno v:
Toxicol Appl Pharmacol
miRNAs (miRNA) are essential players regulating gene expression affecting cellular processes contributing to disease development. Dysregulated miRNA expression has been observed in numerous diseases including hepatitis, cardiovascular diseases and ca
Overexpression of hsa-miR-186 induces chromosomal instability in arsenic-exposed human keratinocytes
Autor:
Laila Al-Eryani, Jiguo Wu, Mark A. Doll, Vanessa States, J. Christopher States, Shesh N. Rai, Sandra S. Wise, Ana Paula Ferragut Cardoso
Publikováno v:
Toxicol Appl Pharmacol
The mechanism of arsenic-induced skin carcinogenesis is not yet fully understood. Chromosomal instability contributes to aneuploidy and is a driving force in carcinogenesis. Arsenic causes mitotic arrest and induces aneuploidy. hsa-miR-186 overexpres
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e2a9aaaed24a3eeb3140539eea13bce1
https://europepmc.org/articles/PMC6746570/
https://europepmc.org/articles/PMC6746570/
Publikováno v:
Toxicological Sciences.
Arsenic is a toxic metalloid widely present in the earth's crust, and is a proven human carcinogen. Chronic arsenic exposure mainly through drinking water causes skin, lung, and urinary bladder cancers, and is associated with liver, prostate, and kid
Autor:
Maria Luiza Cotrim Sartor de Oliveira, Merielen Garcia Nascimento e Pontes, Mitscheli Sanches da Rocha, Ana Paula Ferragut Cardoso, Rafaela Marono Fava, João Lauro Viana de Camargo
Publikováno v:
Toxicology. 333:100-106
Diuron [3-(3,4-dichlorophenyl)-1,1-dimethylurea] is a substituted urea herbicide carcinogenic to the rat urinary bladder at high dietary levels. The suggested non-genotoxic mode of action (MOA) of diuron encompasses cytotoxicity and necrosis followed
Autor:
Douglas C. Wolf, João Lauro Viana de Camargo, Ana Paula Ferragut Cardoso, Shadia M. Ihlaseh-Catalano, Rebecca C. Fry, Hongzu Ren, Kathryn A. Bailey
Publikováno v:
Toxicology. 325:21-30
Diuron (3-(3,4-dichlorophenyl)-1,1-dimethylurea) is a substituted urea herbicide that at high dietary levels (2500 ppm) induces rat urinary bladder hyperplasia after 20 weeks of exposure and neoplasia after 2 years. The effects on the urothelium afte