Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Ana L. Maia"'
Autor:
Fernanda C. B. Oliveira, Eduarda J. Bauer, Carolina M. Ribeiro, Sidney A. Pereira, Bruna T. S. Beserra, Simone M. Wajner, Ana L. Maia, Francisco A. R. Neves, Michella S. Coelho, Angelica A. Amato
Publikováno v:
Frontiers in Endocrinology, Vol 12 (2022)
AimsLiraglutide is a long-acting glucagon-like peptide 1 (GLP-1) receptor agonist used as an anti-hyperglycemic agent in type 2 diabetes treatment and recently approved for obesity management. Weight loss is attributed to appetite suppression, but th
Externí odkaz:
https://doaj.org/article/a5fb4e8c288243129b62b81addb7cf7c
Autor:
Clarissa R. Zaitune, Tatiana L. Fonseca, Luciane P. Capelo, Fatima R. Freitas, Eduardo H. Beber, José M. Dora, Charles C. Wang, Manuela Miranda-Rodrigues, Keico O. Nonaka, Ana L. Maia, Cecilia H. A. Gouveia
Publikováno v:
Frontiers in Endocrinology, Vol 10 (2019)
C3H/HeJ (C3H) mice are deficient of type I deiodinase (D1), an enzyme that activates thyroid hormone (TH), converting thyroxine (T4) to triiodothyronine (T3). Nevertheless, C3H mice present normal serum T3 and a gross euthyroid phenotype. To investig
Externí odkaz:
https://doaj.org/article/df5008aa1e0c46a7bd494380dca4c590
Autor:
Mirela J, de Azevedo, Caroline A, Dalmáz, Maria Luiza A, Caramori, Miriam, Pecis, Jorge F, Esteves, Ana L, Maia, Jorge L, Gross
Publikováno v:
Journal of diabetes and its complications. 16(4)
The aim of this study was to analyze the role of ACE gene insertion/deletion (I/D) and PC-1 gene K121Q polymorphisms in the changes of glomerular filtration rate (GFR), urinary albumin excretion rate (UAER), and blood pressure (BP) levels in a cohort
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::e6bf9a4b18c44eb9ac69f0a09f065f27
https://pubmed.ncbi.nlm.nih.gov/12126783
https://pubmed.ncbi.nlm.nih.gov/12126783
Autor:
Jorge L. Gross, Cristiane Leitão, Ticiana da Costa Rodrigues, Eduardo Camargo, Mirela J. de Azevedo, Sandra P. Silveiro, Ana L. Maia, Mauro Czepielewski
Publikováno v:
Endocrinologist; Nov/Dec2002, Vol. 12 Issue 6, p501-503, 3p
Genotype and phenotype landscape of MEN2 in 554 medullary thyroid cancer patients: the BrasMEN study
Autor:
Rui M B Maciel, Cleber P Camacho, Lígia V M Assumpção, Natassia E Bufalo, André L Carvalho, Gisah A de Carvalho, Luciana A Castroneves, Francisco M de Castro Jr, Lucieli Ceolin, Janete M Cerutti, Rossana Corbo, Tânia M B L Ferraz, Carla V Ferreira, M Inez C França, Henrique C R Galvão, Fausto Germano-Neto, Hans Graf, Alexander A L Jorge, Ilda S Kunii, Márcio W Lauria, Vera L G Leal, Susan C Lindsey, Delmar M Lourenço Jr, Léa M Z Maciel, Patrícia K R Magalhães, João R M Martins, M Cecília Martins-Costa, Gláucia M F S Mazeto, Anelise I Impellizzeri, Célia R Nogueira, Edenir I Palmero, Cencita H C N Pessoa, Bibiana Prada, Débora R Siqueira, Maria Sharmila A Sousa, Rodrigo A Toledo, Flávia O F Valente, Fernanda Vaisman, Laura S Ward, Shana S Weber, Rita V Weiss, Ji H Yang, Magnus R Dias-da-Silva, Ana O Hoff, Sergio P A Toledo, Ana L Maia
Publikováno v:
Endocrine Connections, Vol 8, Iss 3, Pp 289-298 (2019)
Multiple endocrine neoplasia type 2 (MEN2) is an autosomal dominant genetic disease caused by RET gene germline mutations that is characterized by medullary thyroid carcinoma (MTC) associated with other endocrine tumors. Several reports have demonstr
Externí odkaz:
https://doaj.org/article/37d0d78e06b24b48bb6c7725c95f14cc