Zobrazeno 1 - 7 of 7 pro vyhledávání: '"Ana C. Torres-Martínez"'
1
TLR2 activation induced by H. pylori LPS promotes the differential expression of claudin-4, -6, -7 and -9 via either STAT3 and ERK1/2 in AGS cells
Autor: Ana C. Torres-Martínez, Christian O. Chavarría-Velázquez, Erika P. Rendón-Huerta, Luis F. Montaño
Publikováno v: Immunobiology. 223:38-48
Gastric carcinogenesis has been associated to H. pylori virulence factors that induce a chronic inflammation process. Lipopolysaccharides play a role in chronic inflammatory responses via TLR2- and TLR4-dependent signaling pathways. Similarly, cellul
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::13944adc34fa1f4343aaf051008aae43
https://doi.org/10.1016/j.imbio.2017.10.016
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2
Claudin-6 enhances cell invasiveness through claudin-1 in AGS human adenocarcinoma gastric cancer cells
Autor: Luis F. Montaño, A.N. Lara-Holguin, J.F. Gallardo-Vera, Erika P. Rendón-Huerta, Ana C. Torres-Martínez
Publikováno v: Experimental Cell Research. 350:226-235
Claudins participate in tissue barrier function. The loss of this barrier is associated to metalloproteases-related extracellular matrix and basal membranes degradation. Claudin-1 is a pro-MMP-2 activator and claudin-6 transfected AGS (AGS-Cld6) cell
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c7eeab49b86af2d3b80af45af4dea5f4
https://doi.org/10.1016/j.yexcr.2016.11.025
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3
Pegylated interferon-α2b and ribavirin decrease claudin-1 and E-cadherin expression in HepG2 and Huh-7.5 cells
Autor: Erika P. Rendón-Huerta, Ana C. Torres-Martínez, Teresa I. Fortoul, Luis F. Montaño, Ana María Rivas-Estilla, Claudia Charles-Niño, Araceli Páez
Publikováno v: Annals of Hepatology, Vol 12, Iss 4, Pp 448-457 (2013)
Background. Hepatitis C virus (HCV) infection usually results in long-term viremia. Entry of HCV into the he-patocyte requires claudin-1, -6, -9 and occludin. The efficacy of Pegylated interferon-α (PEG-IFN) treatment against HCV infection increased
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b781d63c092df775c9276058a17a1913
http://www.sciencedirect.com/science/article/pii/S1665268119313468
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4
Claudin-6, 7, or 9 Overexpression in the Human Gastric Adenocarcinoma Cell Line AGS Increases Its Invasiveness, Migration, and Proliferation Rate
Autor: Berenice Salas-Morales, Erika P. Rendón-Huerta, Ana C. Torres-Martínez, Veronica Elizabeth Zavala-Zendejas, Teresa I. Fortoul, Luis F. Montaño
Publikováno v: Cancer Investigation. 29:1-11
Altered claudin expression is related to metastatic potential, poor prognosis, or tumor recurrence. We analyzed if the overexpression of claudin-6, claudin-7, or claudin-9 in AGS cells altered cell motility, invasiveness, or proliferation rate. Claud
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c3db0f2f56df050d45ea79b7fc339e99
https://doi.org/10.3109/07357907.2010.512594
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5
Neuroprotective effect of the aminoestrogen prolame against impairment of learning and memory skills in rats injected with amyloid-β-25-35 into the hippocampus
Autor: Monserrat Hernandez, Juan M. Fernández-G, Luis F. Montaño, Erika P. Rendón-Huerta, Ana C. Torres-Martínez, Francisca Pérez-Severiano, Daniel Limon, Jorge Guevara, Alfonso Díaz
Publikováno v: European journal of pharmacology. 685(1-3)
Alzheimer's disease (AD) is a neurodegenerative disorder caused by the deposition of the amyloid-beta peptide (Aβ) in senile plaques and cerebral vasculature. Its neurotoxic mechanisms are associated with the generation of oxidative stress and react
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3f608a2082985e682a1dea83c1e05126
https://pubmed.ncbi.nlm.nih.gov/22542664
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