Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Ana C. P. Thirone"'
Autor:
Maria H M Lima, Andréa M Caricilli, Lélia L de Abreu, Eliana P Araújo, Fabiana F Pelegrinelli, Ana C P Thirone, Daniela M Tsukumo, Ana Flávia M Pessoa, Marinilce F dos Santos, Maria A de Moraes, José B C Carvalheira, Lício A Velloso, Mario J A Saad
Publikováno v:
PLoS ONE, Vol 7, Iss 5, p e36974 (2012)
Wound healing is impaired in diabetes mellitus, but the mechanisms involved in this process are virtually unknown. Proteins belonging to the insulin signaling pathway respond to insulin in the skin of rats.The purpose of this study was to investigate
Externí odkaz:
https://doaj.org/article/ff995adceeda4ee897ead84bcf90b6bf
Autor:
András Masszi, Zalán Péterfi, Katalin Szászi, Christopher A. McCulloch, Andras Kapus, Ana C. P. Thirone, Ori D. Rotstein, Attila Sebe, Hiroyasu Nakano, Lingzhi Fan, Istvan Mucsi
Publikováno v:
Molecular Biology of the Cell. 18:1083-1097
Epithelial-mesenchymal-myofibroblast transition (EMT), a key feature in organ fibrosis, is regulated by the state of intercellular contacts. Our recent studies have shown that an initial injury of cell–cell junctions is a prerequisite for transform
Publikováno v:
Acta Physiologica. 187:257-272
Hyperosmotic stress initiates a variety of compensatory and adaptive responses, which either serve to restore near-normal volume or remodel and reinforce the cell structure to withstand the physical challenge. The latter response is brought about by
Autor:
Andras Kapus, Matthew Zulys, Stine F. Pedersen, Ana C. P. Thirone, Katalin Szászi, Ori D. Rotstein, Pam Speight
Publikováno v:
American journal of physiology. Cell physiology. 296(3)
Hyperosmotic stress induces cytoskeleton reorganization and a net increase in cellular F-actin, but the underlying mechanisms are incompletely understood. Whereas de novo F-actin polymerization likely contributes to the actin response, the role of F-
Publikováno v:
The FASEB Journal. 21
Publikováno v:
Diabetes. 55(4)
Many cytokines increase their receptor affinity for Janus kinases (JAKs). Activated JAK binds to signal transducers and activators of transcription, insulin receptor substrates (IRSs), and Shc. Intriguingly, insulin acting through its own receptor ki
Autor:
Ana C P, Thirone, John A, Scarlett, Alessandra L, Gasparetti, Eliana P, Araujo, Maria H L, Lima, Carla R O, Carvalho, Lício A, Velloso, Mario J A, Saad
Publikováno v:
Diabetes. 51(7)
Growth hormone (GH) and IGFs have a long distinguished history in diabetes, with possible participation in the development of renal complications. The implicated effect of GH in diabetic end-stage organ damage may be mediated by growth hormone recept