Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Ana Belén Herrera-Campos"'
Autor:
Daniel Delgado-Bellido, Esteban Zamudio-Martínez, Mónica Fernández-Cortés, Ana Belén Herrera-Campos, Joaquin Olmedo-Pelayo, Carmen Jordán Perez, José Expósito, Enrique de Álava, Ana Teresa Amaral, Francisco O’ Valle, Angel Garcia Diaz, F. J. Oliver
Publikováno v:
Cell Death and Disease, Vol 14, Iss 2, Pp 1-11 (2023)
Abstract Vasculogenic Mimicry (VM) refers to the capacity to form a blood network from aggressive cancer cells in an independent way of endothelial cells, to provide nutrients and oxygen leading to enhanced microenvironment complexity and treatment f
Externí odkaz:
https://doaj.org/article/4b08f40ad789471d99691db03f23e4ae
Autor:
Esteban Zamudio-Martinez, Ana Belén Herrera-Campos, Alberto Muñoz, José Manuel Rodríguez-Vargas, F. Javier Oliver
Publikováno v:
Journal of Experimental & Clinical Cancer Research, Vol 40, Iss 1, Pp 1-15 (2021)
Abstract Tankyrase 1 (TNKS1) and tankyrase 2 (TNKS2) are two homologous proteins that are gaining increasing importance due to their implication in multiple pathways and diseases such as cancer. TNKS1/2 interact with a large variety of substrates thr
Externí odkaz:
https://doaj.org/article/3b1e510960404a79bb244905483cb9ff
Autor:
Ana Belén Herrera-Campos, Esteban Zamudio-Martinez, Daniel Delgado-Bellido, Mónica Fernández-Cortés, Luis M. Montuenga, F. Javier Oliver, Angel Garcia-Diaz
Publikováno v:
Cancers. 14(11)
Hyperoxia is used in order to counteract hypoxia effects in the TME (tumor microenvironment), which are described to boost the malignant tumor phenotype and poor prognosis. The reduction of tumor hypoxic state through the formation of a non-aberrant
Autor:
Daniel Delgado-Bellido, Esteban Zamudio-Martínez, Mónica Fernández-Cortés, Ana Belén Herrera-Campos, Joaquin Olmedo-Pelayo, Carmen Jordán Perez, José Expósito, Enrique de Álava, Ana Teresa Amaral, Francisco O’ Valle, Angel Garcia Diaz, F. J. Oliver
Publikováno v:
SSRN Electronic Journal.
Vasculogenic Mimicry (VM) refers to the capacity to form a blood network from aggressive cancer cells in an independent way of endothelial cells, to provide nutrients and oxygen leading to enhanced microenvironment complexity and treatment failure. I