Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Ana Azevedo-Pouly"'
Autor:
Lais da Silva, Jinmai Jiang, Corey Perkins, Kalina Rosenova Atanasova, Julie K. Bray, Gamze Bulut, Ana Azevedo-Pouly, Martha Campbell-Thompson, Xiaozhi Yang, Hesamedin Hakimjavadi, Srikar Chamala, Ranjala Ratnayake, Raad Z. Gharaibeh, Chenglong Li, Hendrik Luesch, Thomas D. Schmittgen
Publikováno v:
Cell Death Discovery, Vol 8, Iss 1, Pp 1-10 (2022)
Abstract Pancreatic acinar cells display a remarkable degree of plasticity and can dedifferentiate into ductal-like progenitor cells by a process known as acinar ductal metaplasia (ADM). ADM is believed to be one of the earliest precursor lesions tow
Externí odkaz:
https://doaj.org/article/7f68709dca8b439d9401f8575221c64b
Autor:
Ana Azevedo-Pouly, Michael A Hale, Galvin H Swift, Chinh Q Hoang, Tye G Deering, Jumin Xue, Thomas M Wilkie, L Charles Murtaugh, Raymond J MacDonald
Publikováno v:
PLoS ONE, Vol 18, Iss 10, p e0291512 (2023)
Proper maintenance of mature cellular phenotypes is essential for stable physiology, suppression of disease states, and resistance to oncogenic transformation. We describe the transcriptional regulatory roles of four key DNA-binding transcription fac
Externí odkaz:
https://doaj.org/article/b940bbe07ea24e77a078fe2cacccbed6
Autor:
Varsha G. Desai, Ana Azevedo‐Pouly, Vikrant Vijay, Bounleut Phanavanh, Carrie L. Moland, Tao Han, Javier Revollo, Baikuntha Aryal, V. Ashutosh Rao, James C. Fuscoe
Publikováno v:
Journal of applied toxicology : JATREFERENCES.
Preclinical and clinical findings suggest sexual dimorphism in cardiotoxicity induced by a chemotherapeutic drug, doxorubicin (DOX). However, molecular alterations leading to sex-related differential vulnerability of heart to DOX toxicity are not ful
Autor:
Marie Burdine, Ana Azevedo-Pouly, Lauren Appell, Melanie Barker, Zachary Waldrip, Lyle Burdine
Publikováno v:
The Journal of Immunology. 208:55.15-55.15
Antigen-mediated activation of naïve CD8+ T cells and their differentiation into cytotoxic cells is regulated by a series of signaling mechanisms designed to maintain T cell homeostasis and proper immune responses. Given that cytotoxic CD8+ T cells