Zobrazeno 1 - 10
of 35
pro vyhledávání: '"Amy R. Moser"'
Autor:
Linda A. Schuler, Debra E. Rugowski, Ruth Sullivan, Michael P. Shea, Kathleen A. O'Leary, Amy R. Moser
Publikováno v:
Cancer Lett
Prolactin (PRL) cooperates with other factors to orchestrate mammary development and lactation, and is epidemiologically linked to higher risk for breast cancer. However, how PRL collaborates with oncogenes to foster tumorigenesis and influence breas
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4b0a32e9696980071fedd2010520f542
https://europepmc.org/articles/PMC7995738/
https://europepmc.org/articles/PMC7995738/
Autor:
Joseph Grudzinski, Ashley M. Weichmann, Jason Larrabee, Bryan Bednarz, Benjamin Titz, Kevin R. Kozak, Amy R. Moser, Justin J. Jeffery, Kristofer Lange, Marc Longino, Ian Marsh
Publikováno v:
Cancer Biotherapy and Radiopharmaceuticals. 33:87-95
Purpose: Auger electrons emitted by radioisotopes such as (125)I have a high linear energy transfer and short mean-free path in tissue (95% radiochemical purity was established. To estimate CLR 125 in vivo dosimetry and identify dose-limiting organs,
Autor:
Charanjeet Kaur, David J. Beebe, Lauren M. Zasadil, Sean D. Ryan, David J. Guckenberger, Beth A. Weaver, Eric M. C. Britigan, Amy R. Moser
Publikováno v:
Molecular Biology of the Cell
Expression of a truncated allele of the Apc tumor suppressor causes intestinal tumors with a low rate of chromosomal instability (CIN). Increasing the rate of CIN suppresses tumor growth without inhibiting tumor initiation in both the small intestine
Autor:
Felicite K. Noubissi, Kazuyuki Kawakami, Srikanta Goswami, Yevgenya Grinblat, Toshinari Minamoto, Vladimir S. Spiegelman, Nicholas A. Sanek, Amy R. Moser
Publikováno v:
Cancer Research. 69:8572-8578
金沢大学がん研究所分子標的がん医療研究開発センター
Wnt and Hedgehog signaling pathways play central roles in embryogenesis, stem cell maintenance, and tumorigenesis. However, the mechanisms by which these two pathways in
Wnt and Hedgehog signaling pathways play central roles in embryogenesis, stem cell maintenance, and tumorigenesis. However, the mechanisms by which these two pathways in
Autor:
Kaleigh A. Suhs, Walter J. Hopkins, Christina Kendziorski, Johny E. Elkahwaji, Amy R. Moser, Paulette M. Briggs
Publikováno v:
The Journal of Infectious Diseases. 199:355-361
Background The C3H/HeJ strain of mice develop severe bladder and kidney infections after intravesical inoculation with uropathogenic E. coli. This susceptibility is genetically determined, but the specific genes involved have not been completely defi
Autor:
Amy Gillgrass, G. Shyamala, David C. Seldin, Andrea Rosner, Amy R. Moser, Carol L. MacLeod, Xin Xu, Robert D. Cardiff, Keiko Miyoshi, Esther Landesman-Bollag
Publikováno v:
The American Journal of Pathology. 161:1087-1097
To study phenotype-genotype correlations, ErbB/Ras pathway tumors (transgenic for ErbB2, c-Neu, mutants of c-Neu, polyomavirus middle T antigene (PyV-mT), Ras, and bi-transgenic for ErbB2/Neu with ErbB3 and with progesterone receptor) from four diffe
Autor:
Daniel G. Petereit, L. Jennifer Hahn, Amy Boylan, Mark A. Ritter, Amy R. Moser, Søren M. Bentzen, Shalini Kanekar
Publikováno v:
Frontiers in Oncology, Vol 3 (2013)
Frontiers in Oncology
Frontiers in Oncology
Purpose: To identify sequence variants of the ataxia telangiectasia mutated (ATM) gene and establish their prevalence rate among American Indian (AI) as compared with non-AI cancer patients. Materials and Methods: DNA was isolated from blood samples
Publikováno v:
Mammalian Genome. 11:1058-1062
B6.129S7-Gtrosa26 (B6.R26) mice carry a LacZ-neoR insertion on Chromosome (Chr) 6, made by promoter trapping with 129 ES cells. Female C57BL/6J Apc Min /+ (B6Min/+) mice are highly susceptible to intestinal tumors and to the induction of mammary tumo
Autor:
Carol A. Midgley, Alex R. Shoemaker, Amy R. Moser, Linda Clipson, Michael A. Newton, William F. Dove
Publikováno v:
Proceedings of the National Academy of Sciences. 95:10826-10831
Previous studies of Min /+ (multiple intestinal neoplasia) mice on a sensitive genetic background, C57BL/6 (B6), showed that adenomas have lost heterozygosity for the germ-line Apc Min mutation in the Apc (adenomatous polyposis coli) gene. We now rep
Publikováno v:
Biochimica et Biophysica Acta (BBA) - Reviews on Cancer. 1332:F25-F48