Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Amy B. Ryan"'
Publikováno v:
Neurobiology of Disease, Vol 24, Iss 2, Pp 419-427 (2006)
Huntingtin, the protein product of the Huntington's disease (HD) gene, is known to interact with the tumor suppressor p53. It has recently been shown that activation of p53 upregulates the level of huntingtin, both in vitro and in vivo, whereas p53 d
Externí odkaz:
https://doaj.org/article/629a14ec820d45deb5da9f6aa4560629
Publikováno v:
International Journal of Pediatric Otorhinolaryngology. 122:165-169
Introduction Dysphonia is a known consequence of premature birth, and is usually associated with endotracheal intubation in the neonatal period or surgical ligation of persistent patent ductus arteriosus. Recently, cases of dysphonia, in the absence
Publikováno v:
Journal of Biological Education. 54:540-547
Enzymatic activity is at the core of biological reactions. Understanding them and the chemical and environmental factors that affect reaction rates is critical for biology students. The laboratory ...
Publikováno v:
Neurobiology of Disease, Vol 24, Iss 2, Pp 419-427 (2006)
Huntingtin, the protein product of the Huntington's disease (HD) gene, is known to interact with the tumor suppressor p53. It has recently been shown that activation of p53 upregulates the level of huntingtin, both in vitro and in vivo, whereas p53 d
Autor:
Heidi Scrable, Amy B. Ryan
Publikováno v:
Molecular Imaging. 3:33-42
Reporter genes can monitor the status and activity of recombinant genomes in a diverse array of organisms, from bacteria and yeast to plants and animals. We have combined luciferase reporter genes with a conditional gene expression system based on re
Autor:
Amy B. Ryan, Heidi Scrable
Publikováno v:
Mechanisms of ageing and development. 129(4)
Loss of function mutations in the p53 tumor suppressor gene predispose mice and humans to cancer, resulting in abbreviated life spans. A dominant mutation in the murine HD gene, similar to mutations that cause Huntington's disease in humans, reverses
Occulocutaneous albinism is caused by mutations in the gene encoding the enzyme tyrosinase. Individuals with this disorder are predisposed to visual system deficits. We determined the critical period during development when tyrosinase expression is e
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6b9adb6e20f1998b2315a91c85a8c20d
https://europepmc.org/articles/PMC6740940/
https://europepmc.org/articles/PMC6740940/