Výsledky vyhledávání - "Ammar Salkini"

Akademický článek

The astrocyte-enriched gene deathstar plays a crucial role in the development, locomotion, and lifespan of D. melanogaster

Autor: Xiaoli Zhang, Dongyu Sun, Kyle Wong, Ammar Salkini, Hadi Najafi, Woo Jae Kim

Publikováno v: Fly, Vol 18, Iss 1 (2024)

The Drosophila melanogaster brain is a complex organ with various cell types, orchestrating the development, physiology, and behaviors of the fly. While each cell type in Drosophila brain is known to express a unique gene set, their complete genetic

Externí odkaz: https://doaj.org/article/a4e3c960c8544524ad7535f82112cf01

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Akademický článek

Targeting CD73 with flavonoids inhibits cancer stem cells and increases lymphocyte infiltration in a triple-negative breast cancer mouse model

Publikováno v: Frontiers in Immunology, Vol 15 (2024)

IntroductionChemotherapy remains the mainstay treatment for triple-negative breast cancer (TNBC) due to the lack of specific targets. Given a modest response of immune checkpoint inhibitors in TNBC patients, improving immunotherapy is an urgent and c

Externí odkaz: https://doaj.org/article/10213cd0c20d41adb4e2783bba54bf80

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Astrocyte-like glia-specific gene deathstar is crucial for normal development, adult locomotion and lifespan of male Drosophila

Autor: Hadi Najafi, Kyle Wong, Ammar Salkini, Hongyu Miao, Woo Jae Kim

Background Drosophila melanogaster is a proper model organism for studying the development and function of the nervous system. The Drosophila nervous system consists of distinct cell types with significant homologies to various cell types of more adv

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e11a9f1fd05d29d6e6711fb3830823f3
https://doi.org/10.21203/rs.2.19590/v2

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Abstract P045: Preclinical transcriptome-based evaluation of the translatable potential of new treatments in Triple-Negative Breast Cancer

Autor: Ammar Salkini

Publikováno v: Cancer Immunology Research. 10:P045-P045

Objective: This study will evaluate the potential of four recently proposed TNBC treatments—which all successfully reduced tumor viability in vitro and/or in vivo—to inhibit genes involved in CSC survival, metastatic metabolomic signature, and tu

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::647c5610770d85c0dc2a20f9f56ea6b8
https://doi.org/10.1158/2326-6074.tumimm21-p045

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