Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Amita Rachakonda"'
Autor:
Ning Wang, Gerald D Frank, Ronghua Ding, Zhongjia Tan, Amita Rachakonda, Pier Paolo Pandolfi, Takaaki Senbonmatsu, Erwin J Landon, Tadashi Inagami
Publikováno v:
PLoS ONE, Vol 7, Iss 4, p e35632 (2012)
Pressure overload and prolonged angiotensin II (Ang II) infusion elicit cardiac hypertrophy in Ang II receptor 1 (AT(1)) null mouse, whereas Ang II receptor 2 (AT(2)) gene deletion abolishes the hypertrophic response. The roles and signals of the car
Externí odkaz:
https://doaj.org/article/32c15b54e1cb4800b2ce23088bdabb21
Autor:
Mariam Karamali, Amita Rachakonda, Peggy L. Kendall, Jonathan M. Williams, James W. Thomas, Rachel A. Henry-Bonami
Publikováno v:
The Journal of Immunology. 190:5992-6003
Effective central tolerance is required to control the large extent of autoreactivity normally present in the developing B cell repertoire. Insulin-reactive B cells are required for type 1 diabetes in the NOD mouse, because engineered mice lacking th
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::80fff7162a611a8ceb4b8f7be344a1d0
https://doi.org/10.4049/jimmunol.1201359
https://doi.org/10.4049/jimmunol.1201359
Autor:
Erwin J. Landon, Takaaki Senbonmatsu, Amita Rachakonda, Pier Paolo Pandolfi, Gerald D. Frank, Zhongjia Tan, Tadashi Inagami, Ning Wang, Ronghua Ding
Publikováno v:
PLoS ONE, Vol 7, Iss 4, p e35632 (2012)
PLoS ONE
PLoS ONE
BACKGROUND Pressure overload and prolonged angiotensin II (Ang II) infusion elicit cardiac hypertrophy in Ang II receptor 1 (AT(1)) null mouse, whereas Ang II receptor 2 (AT(2)) gene deletion abolishes the hypertrophic response. The roles and signals
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ee17b7de55e95dc68bc0354efba06bb5
http://europepmc.org/articles/PMC3338737?pdf=render
http://europepmc.org/articles/PMC3338737?pdf=render
Autor:
José A. Terrón, Julius Benicky, Jaroslav Pavel, Amita Rachakonda, Alicia Falcón-Neri, Tadashi Inagami, Juan M. Saavedra
To clarify the relationship between Angiotensin II AT(1) and AT(2) receptors, we studied AT(1) receptor mRNA and binding expression in tissues from AT(2) receptor gene disrupted (AT(2)(-/-)) female mice, where AT(2) receptors are not expressed in viv
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e7752aaf98ad1e77cd30c601e1e32f6e
https://europepmc.org/articles/PMC2789427/
https://europepmc.org/articles/PMC2789427/
Publikováno v:
The Journal of Immunology. 194:139.18-139.18
Immune tolerance breakdown precedes autoimmune disease. Receptor editing is an immune tolerance mechanism that alters BCR specificity to combat frequent autoreactivity in the developing B cell repertoire. Much of what is known about receptor editing
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::4fd9dd635c77bba5e35f87b0854b5fc7
https://doi.org/10.4049/jimmunol.194.supp.139.18
https://doi.org/10.4049/jimmunol.194.supp.139.18