Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Amit Goldberg"'
Autor:
Quan Chen, Amit Goldberg, Reshma Ramlal, Roger A. Fleischman, Hayder Saeed, Bin Huang, Gerhard C. Hildebrandt, Zin W. Myint, Runa Shrestha, Roger H. Herzig, Gregory Monohan
Publikováno v:
Blood. 132:4836-4836
Introduction: Low socioeconomic status (SES) has been shown to shorten survival in classical Hodgkin lymphoma (cHL). We sought to determine if differences in outcome of cHL varied according to SES and geographic setting in a population-based analysis
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::17963958adf08b986d0fca6a00b7ccfa
https://doi.org/10.1182/blood-2018-99-110988
https://doi.org/10.1182/blood-2018-99-110988
Publikováno v:
Experimental hematology. 34(12)
The current study quantified the growth factors in megakaryocytes and monocytes and correlated them to the degree of fibrosis, as there is no quantitative analysis of growth factors from megakaryocytes or monocytes reported in patients with agnogenic
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b448f0ee226b4719c52d37c374e894ea
https://pubmed.ncbi.nlm.nih.gov/17157157
https://pubmed.ncbi.nlm.nih.gov/17157157
Autor:
Kirugaval C. Hemavathy, William Charles, Sramila Aithal, Tsong H. Chang, Jen C. Wang, Haowei Zhang, Allan D. Novetsky, Amit Goldberg
Publikováno v:
Leukemia research. 30(1)
Agnogenic myeloid metaplasia (AMM) is characterized by bone marrow fibrosis and enhanced proliferation of megakaryocytes and CD34+ cells. We have analyzed the factors that could lead to reduced expression of TGF beta1RII in CD34+ cells of AMM patient
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::06bc0394cb8d3da973e7bbf30a2505e9
https://pubmed.ncbi.nlm.nih.gov/16054691
https://pubmed.ncbi.nlm.nih.gov/16054691
Autor:
Amit Goldberg, Jeffrey H. Lipton, Allan D. Novetsky, Steven Lichter, Jen-Chin Wang, Tsong H. Chang
Publikováno v:
Blood. 108:3607-3607
Currently, the prevailing concept concerning the etiology of bone marrow fibrosis in patients with idiopathic myelofibrosis (IMF) is that it results from excessive production of fibrosing growth factors including transforming growth factor beta (TGF-
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::ddca612a2abe021d63fbf064829b81ff
https://doi.org/10.1182/blood.v108.11.3607.3607
https://doi.org/10.1182/blood.v108.11.3607.3607
Publikováno v:
Blood. 106:4272-4272
There are previous reports that growth factors including TGF-β1, PDGF and FGF produced by megakaryocytes are responsible for the etiology of fibrosis in agnogenic myeloid metaplasia (AMM). Due to the technical difficulty in isolating enough megakary
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::9e244dcf5b424d85f6aafbc0d428fdb1
https://doi.org/10.1182/blood.v106.11.4272.4272
https://doi.org/10.1182/blood.v106.11.4272.4272
Autor:
Jen-Chin Wang, Jeffrey Lipton, Tsong S. Chang, Amit Goldberg, Kirugaval C. Hemavathy, Allan D. Novetsky
Publikováno v:
Blood. 104:4749-4749
Growth factors including Transforming Growth Factor-β 1 ( TGF- β1), Platelet Derived Growth Factor (PDGF) and Fibroblast Growth Factor (FGF) have been implicated as responsible for bone marrow fibrosis in AMM. Although TGF-β1 and FGF have been dem
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::146948239f5ebab82092506bd4fcc491
https://doi.org/10.1182/blood.v104.11.4749.4749
https://doi.org/10.1182/blood.v104.11.4749.4749