Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Amelie, Reigl"'
Autor:
Denis Hepbasli, Sina Gredy, Melanie Ullrich, Amelie Reigl, Marco Abeßer, Thomas Raabe, Kai Schuh
Publikováno v:
Brain Sciences, Vol 11, Iss 10, p 1365 (2021)
Vocalization is an important part of social communication, not only for humans but also for mice. Here, we show in a mouse model that functional deficiency of Sprouty-related EVH1 domain-containing 2 (SPRED2), a protein ubiquitously expressed in the
Externí odkaz:
https://doaj.org/article/61961403a09145818af2c4c1b4cfc5f3
Autor:
Verena Schneider, Daniel Kruse, Ives Bernardelli de Mattos, Saskia Zöphel, Kendra-Kathrin Tiltmann, Amelie Reigl, Sarah Khan, Martin Funk, Karl Bodenschatz, Florian Groeber-Becker
Publikováno v:
Biomedicines, Vol 9, Iss 9, p 1153 (2021)
Burns affect millions every year and a model to mimic the pathophysiology of such injuries in detail is required to better understand regeneration. The current gold standard for studying burn wounds are animal models, which are under criticism due to
Externí odkaz:
https://doaj.org/article/c00494e31a8a470ea0f6a2a84711fdde
Gastrointestinal dysfunction in autism displayed by altered motility and achalasia in Foxp1 +/− mice
Autor:
Manuel Stuhlinger, E Zizer, Beate Niesler, Dieter Groneberg, Henning Fröhlich, Gudrun A. Rappold, Marie Luise Kollmeyer, Amelie Reigl, Valerie Catherine Linz, Andreas Friebe
Publikováno v:
Proceedings of the National Academy of Sciences. 116:22237-22245
Gastrointestinal dysfunctions in individuals with autism spectrum disorder are poorly understood, although they are common among this group of patients. FOXP1 haploinsufficiency is characterized by autistic behavior, language impairment, and intellec
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::ada58822b6e916655f9037e4c61522fe
https://doi.org/10.1073/pnas.1911429116
https://doi.org/10.1073/pnas.1911429116
Autor:
Amelie Reigl, Sarah Khan, M Funk, Ives Bernardelli de Mattos, Karl Bodenschatz, Kendra-Kathrin Tiltmann, Verena Schneider, Daniel Kruse, Saskia Zöphel, Florian Groeber-Becker
Publikováno v:
Biomedicines
Biomedicines; Volume 9; Issue 9; Pages: 1153
Biomedicines, Vol 9, Iss 1153, p 1153 (2021)
Biomedicines; Volume 9; Issue 9; Pages: 1153
Biomedicines, Vol 9, Iss 1153, p 1153 (2021)
Burns affect millions every year and a model to mimic the pathophysiology of such injuries in detail is required to better understand regeneration. The current gold standard for studying burn wounds are animal models, which are under criticism due to
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ae69b0ab327e6f50aa24941742c3406f
https://publica.fraunhofer.de/handle/publica/270023
https://publica.fraunhofer.de/handle/publica/270023
Autor:
Henning, Fröhlich, Marie Luise, Kollmeyer, Valerie Catherine, Linz, Manuel, Stuhlinger, Dieter, Groneberg, Amelie, Reigl, Eugen, Zizer, Andreas, Friebe, Beate, Niesler, Gudrun, Rappold
Publikováno v:
Proceedings of the National Academy of Sciences of the United States of America. 116(44)
Gastrointestinal dysfunctions in individuals with autism spectrum disorder are poorly understood, although they are common among this group of patients. FOXP1 haploinsufficiency is characterized by autistic behavior, language impairment, and intellec
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::fb53bcee8192d68e338a4f2fe2838bad
https://pubmed.ncbi.nlm.nih.gov/31611379
https://pubmed.ncbi.nlm.nih.gov/31611379
Autor:
Alex Vincent, Barbara Voussen, Amelie Reigl, Jan D. Huizinga, Sean P. Parsons, Katharina Beck, Andreas Friebe
Publikováno v:
Neurogastroenterology and motility : the official journal of the European Gastrointestinal Motility Society. 31(6)
Background Nitric oxide (NO) mediates inhibitory neurotransmission and is a critical component of neuronal programs that generate propulsive contractions. NO acts via its receptor NO-sensitive guanylyl cyclase (NO-GC) which is expressed in smooth mus
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::11e6e2a280250917d87d15d7e3896ec5
https://pubmed.ncbi.nlm.nih.gov/30947401
https://pubmed.ncbi.nlm.nih.gov/30947401