Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Amanda Jane Zatta"'
Autor:
Jeremiah Deneve, Rong Jiang, Zhi-Qing Zhao, James Mykytenko, Hajime Kin, James G. Reeves, Ning-Ping Wang, Amanda Jane Zatta, Jakob Vinten-Johansen, Robert A. Guyton
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 293:H2845-H2852
Protease-activated receptor-2 (PAR-2) may have proinflammatory effects in some tissues and protective effects in other tissues. The role of PAR-2 in in vivo myocardial ischemia-reperfusion has not yet been determined. This study tested the hypothesis
Autor:
Rong Jiang, Zhi-Qing Zhao, Michael E. Halkos, Hajime Kin, Jakob Vinten-Johansen, Faraz Kerendi, Amanda Jane Zatta, Robert A. Guyton
Publikováno v:
Basic Research in Cardiology. 100:404-412
OBJECTIVES A series of brief coronary artery reperfusions and reocclusions applied during the early minutes of coronary artery reflow ("postconditioning") attenuates reperfusion injury. However, it is not known whether brief ischemia-reperfusion appl
Autor:
Amanda Jane Zatta, Nikita Lee Schembri, Louise Elizabeth Phillips, James P. Isbister, Alice Noone
Publikováno v:
Current drug targets. 10(8)
Recombinant activated factor VII (rFVIIa), developed and effective in managing inhibitors in haemophilia patients, is being widely used off-label as a "panhaemostatic agent" with ongoing controversy as to its benefits and risks in terms of controllin
Autor:
Zhi Qing Zhao, Jeremiah Deneve, James Mykytenko, Amanda Jane Zatta, Ning Ping Wang, Rong Jiang, James G. Reeves, Hajime Kin, Robert A. Guyton, Jakob Vinten-Johansen
Publikováno v:
Shock (Augusta, Ga.). 29(6)
Oxidative stress-stimulated nuclear factor-kappa B (NF-kappa B) activation has been associated with rapid transcription of TNF-alpha and induction of apoptosis. This study tested the hypothesis that postconditioning (Postcon) reduces myocardial apopt
Publikováno v:
Journal of applied physiology (Bethesda, Md. : 1985). 103(4)
Reperfusion is the definitive treatment to salvage ischemic myocardium from infarction. A primary determinant of infarct size is the duration of ischemia. In myocardium that has not been irreversibly injured by ischemia, reperfusion induces additiona