Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Amanda Indalécio Goulart"'
Autor:
Diogo Dominguini, Amanda Indalécio Goulart, Monique Michels, Felipe Dal-Pizzol, Filipe Fernandes, Heloisa Borges, Cristiane Ritter, Mariane Rocha, Andriele Vieira
Publikováno v:
Neurochemistry international. 142
The nervous system is one of the first systems to be affected during sepsis. Sepsis not only has a high risk of mortality, but could also lead to cerebral dysfunction and cognitive impairment in long-term survival patients. The receptor for advanced
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b14ca2880f12c7706fc893dedd814a9b
https://pubmed.ncbi.nlm.nih.gov/33232757
https://pubmed.ncbi.nlm.nih.gov/33232757
Autor:
Pricila Ávila, Mariane Abatti, Diogo Dominguini, Monique Michels, Emily Córneo, Heloisa Borges, Felipe Dal-Pizzol, Tatiana Barichello, Andriele Vieira, Amanda Indalécio Goulart
Publikováno v:
Clinical science (London, England : 1979). 134(7)
Background: In order to modulate microglial phenotypes in vivo, M1 microglia were depleted by administration of gadolinium chloride and the expression of M2 microglia was induced by IL-4 administration in an animal model of sepsis to better character
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5623ebf2dae06e8843d55d589c4fecf3
https://pubmed.ncbi.nlm.nih.gov/32633321
https://pubmed.ncbi.nlm.nih.gov/32633321
Autor:
Heloisa Borges, Tatiana Barichello, Bruna Pescador, Pricila Ávila, Andriele Vieira, Luana Cucker, Felipe Dal-Pizzol, Mariane Abatti, Celso Carvalho Junior, Amanda Indalécio Goulart, Monique Michels, João Quevedo
Publikováno v:
Molecular neurobiology. 56(11)
Sepsis-associated encephalopathy is highly prevalent and has impact both in early and late morbidity and mortality. The mechanisms by which sepsis induces brain dysfunction include neuroinflammation, disrupted blood–brain barrier, oxidative stress,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::af8166ef84f3898116f57882cd776a84
https://pubmed.ncbi.nlm.nih.gov/31020614
https://pubmed.ncbi.nlm.nih.gov/31020614