Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Amanda Beauchamp"'
Autor:
Yen Yi Ho, Lin Zhang, Andrew DiLernia, Amanda Beauchamp, Lisa A. Peterson, Igor V. Ignatovich, Alex E. Grill
Publikováno v:
Chem Res Toxicol
Metabolic activation of many carcinogens leads to formation of reactive intermediates that form DNA adducts. These adducts are cytotoxic when they interfere with cell division. They can also cause mutations by miscoding during DNA replication. Theref
Autor:
Amanda Beauchamp
Publikováno v:
Journal of Media Practice. 12:235-243
This article draws upon Karen Lury's definitions of 'space' and 'place' in relation to the BBC children's programme Blue Peter (1958–present). Through an analysis of the Blue Peter studio over the past 53 years, Amanda Beauchamp highlights its evol
Autor:
Amanda Beauchamp, Jill Wykosky, Denise M. Gibo, Akiva Mintz, Mark O. Lively, Waldemar Debinski
EphrinA1 is a glycosylphosphatidylinositol (GPI)-linked ligand for the EphA2 receptor, which is overexpressed in glioblastoma (GBM), among other cancers. Activation of the receptor by ephrinA1 leads to a suppression of oncogenic properties of GBM cel
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::53f0b975722ffd6199a78761f7e4d225
https://europepmc.org/articles/PMC3434542/
https://europepmc.org/articles/PMC3434542/
Autor:
Amanda Beauchamp, Waldemar Debinski
Ephrin-A1 and its primary receptor, EphA2, are involved in numerous physiological processes and have been intensely studied for their roles in malignancy. Ephrin-Eph signalling is complex on its own and is also cell-type dependent, making elucidation
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6c44f6c3b1df7464dadb864667d4796c
https://europepmc.org/articles/PMC3288643/
https://europepmc.org/articles/PMC3288643/
Publikováno v:
Cancer Research. 71:1218-1218
Introduction. EphrinA1 is a GPI-linked ligand for the EphA2 receptor, which is overexpressed in Glioblastoma Multiforme (GBM). Activation of the receptor by ephrinA1 leads to a decrease in the oncogenic properties of GBM cells. We have recently uncov
Autor:
Tiara Murphy, Van Nguyen, Roy R. Hantgan, Amanda Beauchamp, Waldemar Debinski, Akiva Mintz, Jesse M. Conyers
Publikováno v:
Cancer Research. 71:1650-1650
Our goal is to therapeutically exploit Glioblastoma Multiforme (GBM) restricted biomarkers that are not expressed in normal brain. Since GBMs are highly heterogeneous tumors, targeting any single biomarker will likely not be relevant to all GBMs. Thu