Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Amal Kouadri"'
Copper-Associated Oxidative Stress Contributes to Cellular Inflammatory Responses in Cystic Fibrosis
Autor:
Amal Kouadri, Johanna Cormenier, Kevin Gemy, Laurence Macari, Peggy Charbonnier, Pierre Richaud, Isabelle Michaud-Soret, Nadia Alfaidy, Mohamed Benharouga
Publikováno v:
Biomedicines, Vol 9, Iss 4, p 329 (2021)
Cystic fibrosis (CF) is caused by mutations in the gene encoding the CF Transmembrane Conductance Regulator (CFTR), an apical chloride channel. An early inflammation (EI) in the lung of CF patients occurring in the absence of any bacterial infection
Externí odkaz:
https://doaj.org/article/40c21c30a05c4b91a3b2ffa7bfe5f3c0
Autor:
Pierre Richaud, Mariam El Khatib, Sylvain Chauvet, Micheline El Khoury, Johanna Cormenier, Christelle Coraux, Wael Zeinyeh, Mohamed Benharouga, Laurence Macari, Nadia Alfaidy, Isabelle Michaud-Soret, Amal Kouadri
Publikováno v:
Antioxidants and Redox Signaling
Antioxidants and Redox Signaling, 2019, 31 (1), pp.59-74. ⟨10.1089/ars.2018.7500⟩
Antioxidants and Redox Signaling, Mary Ann Liebert, 2019, 31 (1), pp.59-74. ⟨10.1089/ars.2018.7500⟩
Antioxidants and Redox Signaling, 2019, 31 (1), pp.59-74. ⟨10.1089/ars.2018.7500⟩
Antioxidants and Redox Signaling, Mary Ann Liebert, 2019, 31 (1), pp.59-74. ⟨10.1089/ars.2018.7500⟩
International audience; AIM: Bronchial epithelium acts as a defensive barrier against inhaled pollutants and microorganisms. This barrier is often compromised in inflammatory airway diseases that are characterized by excessive oxidative stress respon
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::178a063d0c24f289cf9bed60489440a8
https://hal.science/hal-02092968
https://hal.science/hal-02092968
Autor:
Mohamed Benharouga, Wael Traboulsi, Laura Thevenon, Nadia Alfaidy, Boubou Camara, Sylvain Chauvet, Amal Kouadri, Jean-Jacques Feige
Publikováno v:
American Journal of Physiology-Lung Cellular and Molecular Physiology
American Journal of Physiology-Lung Cellular and Molecular Physiology, American Physiological Society, 2015, 309 (3), pp.L314-L322. ⟨10.1152/ajplung.00382.2014⟩
American Journal of Physiology-Lung Cellular and Molecular Physiology, 2015, 309 (3), pp.L314-L322. ⟨10.1152/ajplung.00382.2014⟩
American Journal of Physiology-Lung Cellular and Molecular Physiology, American Physiological Society, 2015, 309 (3), pp.L314-L322. ⟨10.1152/ajplung.00382.2014⟩
American Journal of Physiology-Lung Cellular and Molecular Physiology, 2015, 309 (3), pp.L314-L322. ⟨10.1152/ajplung.00382.2014⟩
Enhanced lung angiogenesis has been reported in cystic fibrosis (CF). Recently, two highly homologous ligands, endocrine gland vascular endothelial growth factor (EG-VEGF) and mammalian Bv8, have been described as new angiogenic factors. Both ligands
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::35c166ae8a0ac02830519a9953ad65f6
https://hal.archives-ouvertes.fr/hal-01207814
https://hal.archives-ouvertes.fr/hal-01207814