Zobrazeno 1 - 10
of 56
pro vyhledávání: '"Altan Rentsendorj"'
Autor:
Ron Danziger, Dieu-Trang Fuchs, Yosef Koronyo, Altan Rentsendorj, Julia Sheyn, Eric Y. Hayden, David B. Teplow, Keith L. Black, Sebastien Fuchs, Kenneth E. Bernstein, Maya Koronyo-Hamaoui
Publikováno v:
Frontiers in Physiology, Vol 14 (2023)
This review examines the role of angiotensin-converting enzyme (ACE) in the context of Alzheimer’s disease (AD) and its potential therapeutic value. ACE is known to degrade the neurotoxic 42-residue long alloform of amyloid β-protein (Aβ42), a pe
Externí odkaz:
https://doaj.org/article/b54ad841e64e40d8a609a58081f4ca5b
Autor:
Altan Rentsendorj, Koen Raedschelders, Dieu-Trang Fuchs, Julia Sheyn, Vineet Vaibhav, Rebecca A. Porritt, Haoshen Shi, Jargalsaikhan Dagvadorj, Juliana de Freitas Germano, Yosef Koronyo, Moshe Arditi, Keith L. Black, Bhakta Prasad Gaire, Jennifer E. Van Eyk, Maya Koronyo-Hamaoui
Publikováno v:
Frontiers in Immunology, Vol 14 (2023)
IntroductionOsteopontin (OPN; also known as SPP1), an immunomodulatory cytokine highly expressed in bone marrow-derived macrophages (BMMΦ), is known to regulate diverse cellular and molecular immune responses. We previously revealed that glatiramer
Externí odkaz:
https://doaj.org/article/0841f2b649ca4207b40888b9d0da4ad7
Autor:
Haoshen Shi, Yosef Koronyo, Altan Rentsendorj, Dieu-Trang Fuchs, Julia Sheyn, Keith L. Black, Nazanin Mirzaei, Maya Koronyo-Hamaoui
Publikováno v:
Frontiers in Neuroscience, Vol 15 (2021)
The retina has been increasingly investigated as a site of Alzheimer’s disease (AD) manifestation for over a decade. Early reports documented degeneration of retinal ganglion cells and their axonal projections. Our group provided the first evidence
Externí odkaz:
https://doaj.org/article/751f43151f20454eadfd239f4d763558
Autor:
Arielle Kasindi, Dieu-Trang Fuchs, Yosef Koronyo, Altan Rentsendorj, Keith L. Black, Maya Koronyo-Hamaoui
Publikováno v:
Cells, Vol 11, Iss 9, p 1578 (2022)
Novel, neuroprotective uses of Copaxone (generic name: glatiramer acetate—GA) are being examined, primarily in neurological conditions involving cognitive decline. GA is a well-studied synthetic copolymer that is FDA-approved for immune-based treat
Externí odkaz:
https://doaj.org/article/b5844253154f46f0862f7d1be3405d4a
Autor:
Nazanin Mirzaei, Haoshen Shi, Mia Oviatt, Jonah Doustar, Altan Rentsendorj, Dieu-Trang Fuchs, Julia Sheyn, Keith L. Black, Yosef Koronyo, Maya Koronyo-Hamaoui
Publikováno v:
Frontiers in Neuroscience, Vol 14 (2020)
The neurosensory retina emerges as a prominent site of Alzheimer’s disease (AD) pathology. As a CNS extension of the brain, the neuro retina is easily accessible for noninvasive, high-resolution imaging. Studies have shown that along with cognitive
Externí odkaz:
https://doaj.org/article/73e54d03b5684d3b807fbea2ccdd215d
Autor:
Leah R. Zuroff, Tania Torbati, Nadav J. Hart, Dieu-Trang Fuchs, Julia Sheyn, Altan Rentsendorj, Yosef Koronyo, Eric Y. Hayden, David B. Teplow, Keith L. Black, Maya Koronyo-Hamaoui
Publikováno v:
Frontiers in Immunology, Vol 11 (2020)
Interleukin-34 (IL-34) is a recently discovered cytokine that acts as a second ligand of the colony stimulating factor 1 receptor (CSF1R) in addition to macrophage colony-stimulating factor (M-CSF). Similar to M-CSF, IL-34 also stimulates bone marrow
Externí odkaz:
https://doaj.org/article/dcf8c0e6c7a843eaab90cca739b5a855
Autor:
Songlin Li, Eric Y. Hayden, Veronica J. Garcia, Dieu-Trang Fuchs, Julia Sheyn, David A. Daley, Altan Rentsendorj, Tania Torbati, Keith L. Black, Ueli Rutishauser, David B. Teplow, Yosef Koronyo, Maya Koronyo-Hamaoui
Publikováno v:
Frontiers in Immunology, Vol 11 (2020)
Impaired synaptic integrity and function due to accumulation of amyloid β-protein (Aβ42) oligomers is thought to be a major contributor to cognitive decline in Alzheimer's disease (AD). However, the exact role of Aβ42 oligomers in synaptotoxicity
Externí odkaz:
https://doaj.org/article/392670f2dede4919b7ce7150eba292cf
Autor:
Lincoln A. Edwards, Aiguo Li, Dror Berel, Mecca Madany, Nam-Ho Kim, Minzhi Liu, Mitch Hymowitz, Benjamin Uy, Rachel Jung, Minlin Xu, Keith L. Black, Altan Rentsendorj, Xuemo Fan, Wei Zhang, John S. Yu
Publikováno v:
Scientific Reports, Vol 7, Iss 1, Pp 1-11 (2017)
Abstract The identification of a stem cell regulatory gene which is aberrantly expressed in glioma and associated with patient survival would increase the understanding of the role of glioma cancer stem cells (GCSCs) in the virulence of gliomas. Inte
Externí odkaz:
https://doaj.org/article/20170381f56644fd9638ce225777a1a9
Autor:
Yosef Koronyo, Altan Rentsendorj, Nazanin Mirzaei, Giovanna C. Regis, Julia Sheyn, Haoshen Shi, Ernesto Barron, Galen Cook-Wiens, Anthony R. Rodriguez, Rodrigo Medeiros, Joao A. Paulo, Veer B. Gupta, Andrei A. Kramerov, Alexander V. Ljubimov, Jennifer E. Van Eyk, Stuart L. Graham, Vivek K. Gupta, John M. Ringman, David R. Hinton, Carol A. Miller, Keith L. Black, Antonino Cattaneo, Giovanni Meli, Mehdi Mirzaei, Dieu-Trang Fuchs, Maya Koronyo-Hamaoui
Publikováno v:
Acta Neuropathologica. 145:409-438
Alzheimer’s disease (AD) pathologies were discovered in the accessible neurosensory retina. However, their exact nature and topographical distribution, particularly in the early stages of functional impairment, and how they relate to disease progre
Autor:
Yosef Koronyo, Altan Rentsendorj, Nazanin Mirzaei, Giovanna C. Regis, Julia Sheyn, Haoshen Shi, Ernesto Barron, Galen Cook-Wiens, Anthony R. Rodriguez, Rodrigo Medeiros, Joao A. Paulo, Veer B. Gupta, Andrei A. Kramerov, Alexander V. Ljubimov, Jennifer E. Van Eyk, Stuart L. Graham, Vivek K. Gupta, John M. Ringman, David R. Hinton, Carol A. Miller, Keith L. Black, Antonino Cattaneo, Giovanni Meli, Mehdi Mirzaei, Dieu-Trang Fuchs, Maya Koronyo-Hamaoui
Alzheimer’s disease (AD) pathologies were discovered in the easily accessible neurosensory retina. Yet, their specific nature, topographical distribution, and relationship with disease status remain undefined. Here, we histologically determined bur
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::ec03b24209d33ac620b6e6ff6dbc08bf
https://doi.org/10.21203/rs.3.rs-2152218/v1
https://doi.org/10.21203/rs.3.rs-2152218/v1