Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Alistair Colin Church"'
Autor:
Michael McGettrick, Helen Dormand, Melanie Brewis, Martin K. Johnson, Ninian N. Lang, Alistair Colin Church
Publikováno v:
Frontiers in Cardiovascular Medicine, Vol 9 (2022)
BackgroundVentricular septal flattening reflects RV pressure overload in pulmonary arterial hypertension. Eccentricity index (EI) and pulmonary artery distensibility (PAD) correlate with pulmonary artery pressure. We assessed the utility of these usi
Externí odkaz:
https://doaj.org/article/338e2c798596435db8e178aa069df77e
Autor:
Ninian N Lang, Michael McGettrick, Catherine Bagot, M K Johnson, Alistair Colin Church, Alexander MacLellan, Paul McCaughey, Melanie J Brewis
Publikováno v:
BMJ Open, Vol 11, Iss 8 (2021)
Externí odkaz:
https://doaj.org/article/aaa3715c3507430780b17c3f4f89a558
Autor:
Luke Lawson McLean, Katherine Pellino, Melanie Brewis, Andrew Peacock, Martin Johnson, Alistair Colin Church
Publikováno v:
ERJ Open Research, Vol 5, Iss 4 (2019)
Externí odkaz:
https://doaj.org/article/d7d883c1b5904aa6b1b364e906ab89c9
Autor:
Robina Kate Coker, Alison Armstrong, Alistair Colin Church, Steve Holmes, Jonathan Naylor, Katharine Pike, Peter Saunders, Kristofer John Spurling, Pamela Vaughn
Publikováno v:
Thorax. 77(4)
Autor:
Martin Johnson, Andrew J. Peacock, Katherine Pellino, Melanie J. Brewis, Luke Lawson McLean, Alistair Colin Church
Publikováno v:
ERJ Open Research
ERJ Open Research, Vol 5, Iss 4 (2019)
ERJ Open Research, Vol 5, Iss 4 (2019)
Pulmonary arterial hypertension (PAH) is a rare disease characterised by progressive pulmonary vascular remodelling, resulting in right ventricular failure, which is the principal cause of death [1]. Excessive bodyweight is a well-established indepen
Publikováno v:
Thorax. 67:258-262
Autor:
Alistair Colin Church, Gareth Bryson, Andrew J. Peacock, Damien H. Martin, Andrew J. Fisher, David J. Welsh, Roger M. Wadsworth
Publikováno v:
American Journal of Physiology-Lung Cellular and Molecular Physiology
The p38 mitogen-activated protein kinase (MAPK) system is increasingly recognized as an important inflammatory pathway in systemic vascular disease but its role in pulmonary vascular disease is unclear. Previous in vitro studies suggest p38 MAPKα is