Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Alireza Saadatmand"'
Publikováno v:
Journal of Public Transportation, Vol 25, Iss , Pp 100072- (2023)
Planning public transport highly relies on the availability, quantity and quality of travel demand data of passengers. In the last two decades, smart card data has provided the opportunity to create comprehensive travel demand data as a byproduct of
Externí odkaz:
https://doaj.org/article/e0becbd98f9e4d06bb633a560399c1a9
Autor:
Jakob Morgenstern, Sylvia Katz, Jutta Krebs-Haupenthal, Jessy Chen, Alireza Saadatmand, Fabiola Garcia Cortizo, Alexandra Moraru, Johanna Zemva, Marta Campos Campos, Aurelio Teleman, Johannes Backs, Peter Nawroth, Thomas Fleming
Publikováno v:
Cell Reports, Vol 32, Iss 12, Pp 108160- (2020)
Summary: The glyoxalase system is a highly conserved and ubiquitously expressed enzyme system, which is responsible for the detoxification of methylglyoxal (MG), a spontaneous by-product of energy metabolism. This study is able to show that a phospho
Externí odkaz:
https://doaj.org/article/e60809eadc724749a098946747791b97
Autor:
Hamid Kalantari, Bahram Bagherpour, Tahmine Tavakoli, Mahsa Khodadoostan, Seyed Mehdi Hejazi, Alireza Saadatmand
Publikováno v:
Journal of Research in Medical Sciences, Vol 24, Iss 1, Pp 12-12 (2019)
Background: The present study aimed to determine the frequency of the IL28B polymorphism rs8099917 in patients with genotype 1 hepatitis C virus (HCV) infection treated with pegylated-interferon-α2b (PEG-IFN-α2b) and ribavirin (RBV) and its treatme
Externí odkaz:
https://doaj.org/article/639f14c4dd534f369b5e400f9315996b
Autor:
Alireza Saadatmand, Mark Pepin, Uwe Haberkorn, Jürgen Okun, Lisa Schlicker, Almut Schulze, Johannes Backs
Publikováno v:
Journal of Molecular and Cellular Cardiology. 173:189
Publikováno v:
Circulation. 144
Introduction: Ca 2+ /calmodulin-dependent protein kinase II (CaMK2) is a recognized driver of pathological cardiac remodeling and heart failure, but its role in the metabolic reprogramming of heart failure remains unclear, as do the molecular process
Autor:
Aurelio A. Teleman, Jessy Chen, Peter P. Nawroth, Thomas Fleming, Johanna Zemva, Marta Campos Campos, Johannes Backs, Alireza Saadatmand, Fabiola Garcia Cortizo, Jakob Morgenstern, Alexandra Moraru, Sylvia Katz, Jutta Krebs-Haupenthal
Publikováno v:
Cell Reports, Vol 32, Iss 12, Pp 108160-(2020)
Summary: The glyoxalase system is a highly conserved and ubiquitously expressed enzyme system, which is responsible for the detoxification of methylglyoxal (MG), a spontaneous by-product of energy metabolism. This study is able to show that a phospho
Autor:
Aurelio A. Teleman, Jutta Krebs-Haupenthal, Peter P. Nawroth, Sylvia Katz, Alexandra Moraru, Jessy Chen, Jakob Morgernstern, Johannes Backs, Thomas Fleming, Alireza Saadatmand, Fabiola Garcia Cortizo, Marta Campos Campos, Johanna Zemva
The glyoxalase system is a ubiquitously expressed enzyme system with narrow substrate specificity and is responsible for the detoxification of harmful methylglyoxal (MG), a spontaneous by-product of energy metabolism. Glyoxalase 1 (Glo1) is the first
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bee6738010b078b90ef0fc0578af5d81
Autor:
Senka Ljubojevic, Steffen Pabel, Gerd Hasenfuß, Theodor Tirilomis, Yansong Wang, André Renner, Ali El-Armouche, Thomas H. Fischer, Silvio Weber, Jan Gummert, Alireza Saadatmand, Jörg Eiringhaus, Samuel Sossalla, Maja Köhn, Nataliya Dybkova, Lars S. Maier
Publikováno v:
European Journal of Heart Failure. 20:1673-1685
Background Disruption of Ca2+ homeostasis is a key pathomechanism in heart failure. CaMKII-dependent hyperphosphorylation of ryanodine receptors in the sarcoplasmic reticulum (SR) increases the arrhythmogenic SR Ca2+ leak and depletes SR Ca2+ stores.
Autor:
Thomas H, Fischer, Jörg, Eiringhaus, Nataliya, Dybkova, Alireza, Saadatmand, Steffen, Pabel, Silvio, Weber, Yansong, Wang, Maja, Köhn, Theodor, Tirilomis, Senka, Ljubojevic, André, Renner, Jan, Gummert, Lars S, Maier, Gerd, Hasenfuß, Ali, El-Armouche, Samuel, Sossalla
Publikováno v:
European journal of heart failure. 20(12)
Disruption of CaHuman myocardium from three groups of patients was investigated: (i) healthy controls (non-failing, NF, n = 8), (ii) compensated hypertrophy (Hy, n = 16), and (iii) end-stage heart failure (HF, n = 52). Expression of PP1 was unchanged