Zobrazeno 1 - 10
of 28
pro vyhledávání: '"Aline M Hilzendeger"'
Autor:
Aline M Hilzendeger, Letícia Antunes Athayde Souza, Mariléia Chaves Andrade, Sérgio Avelino Mota Nobre, Alfredo Maurício Batista de Paula, Deborah de Farias Lelis, Juliana Pinto de Lima, André Luiz Sena Guimarães, Ronize Viviane Jorge Brito, Sérgio Henrique Sousa Santos, Keila Lopes Mendes
Publikováno v:
Protein & Peptide Letters. 28:761-768
Background:: The microbiome is now known for its important role in whole-body homeostasis. A dysbiosis of the normal microbiota is correlated with metabolic disorders. In this sense, the search for compounds able to modulate the microbiome is needed.
Publikováno v:
Protein and peptide letters. 28(7)
Flavonoids are commonly found in fruits, vegetables, and plant-derived foods and may promote various health benefits when included in the diet. The biological activity of flavonoids is normally associated to their potent antioxidant and anti-inflamma
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Autor:
João Bosco Pesquero, Clovis R. Nakaie, Rafael Leite Morais, Michael Bader, Jair R. Chagas, Natalia Alenina, Marcelo A. Mori, Adriana K. Carmona, Aline M Hilzendeger, Mihail Todiras, Bruna Visniauskas, Ronaldo C. Araujo
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 312:H437-H445
Obesity is assumed to be a major cause of human essential hypertension; however, the mechanisms responsible for weight-related increase in blood pressure (BP) are not fully understood. The prevalence of hypertension induced by obesity has grown over
Autor:
Aline M Hilzendeger, Anthony P. Thompson, Justin L. Grobe, Fusakazu Jo, Hiromi Jo, Robin L. Davisson, Martin D. Cassell, Curt D. Sigmund, D. Thomas Rutkowski
Publikováno v:
Hypertension. 65:1341-1348
Endoplasmic reticulum stress has become an important mechanism in hypertension. We examined the role of endoplasmic reticulum stress in mediating the increased saline-intake and hypertensive effects in response to deoxycorticosterone acetate (DOCA)
Autor:
Harald M. Stauss, Aline M Hilzendeger, Curt D. Sigmund, Justin L. Grobe, Allyn L. Mark, Martin D. Cassell, Deborah R. Davis
Publikováno v:
Hypertension. 61:716-722
Although elevated renin–angiotensin system activity and angiotensinergic signaling within the brain are required for hypertension, polydipsia, and increased metabolic rate induced by deoxycorticosterone acetate (DOCA)-salt, the contribution of spec
Autor:
Rafael L, Morais, Aline M, Hilzendeger, Bruna, Visniauskas, Mihail, Todiras, Natalia, Alenina, Marcelo A, Mori, Ronaldo C, Araújo, Clovis R, Nakaie, Jair R, Chagas, Adriana K, Carmona, Michael, Bader, João B, Pesquero
Publikováno v:
American journal of physiology. Heart and circulatory physiology. 312(3)
Obesity is assumed to be a major cause of human essential hypertension; however, the mechanisms responsible for weight-related increase in blood pressure (BP) are not fully understood. The prevalence of hypertension induced by obesity has grown over
Autor:
Aline M Hilzendeger, Xuebo Liu, Leonard A. Brooks, David Dellsperger, Justin L. Grobe, Allyn L. Mark, Kamal Rahmouni, Donald A. Morgan, Curt D. Sigmund
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 303:H197-H206
The sympathetic nervous system, leptin, and renin-angiotensin system (RAS) have been implicated in obesity-associated hypertension. There is increasing evidence for the presence of both leptin and angiotensin II receptors in several key brain cardiov
Autor:
Aline M Hilzendeger, Alberto Navarro, Dulce Elena Casarini, Jorge L. Pesquero, Liliam Fernandes, Marcelo A. Mori, João Bosco Pesquero, Felipe C.G. Reis, Regiane A. Sabatini, Adriana K. Carmona, Maria Claudina Camargo de Andrade, Paola Bianchi Guimarães, Jair R. Chagas, Edson Lucas dos Santos, Patrícia Alessandra Bersanetti, Michael Bader
Publikováno v:
Hypertension. 51:689-695
Angiotensin-converting enzyme (ACE) is an ectoprotein able to modulate the activity of a plethora of compounds, among them angiotensin I and bradykinin. Despite several decades of research, new aspects of the mechanism of action of ACE have been eluc
Autor:
Henry L. Keen, Pimonrat Ketsawatsomkron, Justin L. Grobe, Ko-Ting Lu, Madeliene Stump, Curt D. Sigmund, Deborah R. Davis, Frank M. Faraci, Aline M Hilzendeger, T. Michael De Silva
Publikováno v:
Hypertension (Dallas, Tex. : 1979). 67(1)
Loss of peroxisome proliferator–activated receptor-γ (PPARγ) function causes hypertension, whereas its activation lowers blood pressure. Evidence suggests that these effects may be attributable to PPARγ activity in the vasculature. However, the