Zobrazeno 1 - 10
of 17
pro vyhledávání: '"Alina S. Bilal"'
Autor:
Erik A. Blackwood, Alina S. Bilal, Winston T. Stauffer, Adrian Arrieta, Christopher C. Glembotski
Publikováno v:
Cells, Vol 9, Iss 3, p 602 (2020)
The heart exhibits incredible plasticity in response to both environmental and genetic alterations that affect workload. Over the course of development, or in response to physiological or pathological stimuli, the heart responds to fluctuations in wo
Externí odkaz:
https://doaj.org/article/d86336a4286b4fe28b8930ee947fdb11
Autor:
Erik A. Blackwood, Lauren F. MacDonnell, Donna J. Thuerauf, Alina S. Bilal, Victoria B. Murray, Kenneth C. Bedi, Kenneth B. Margulies, Christopher C. Glembotski
Publikováno v:
Circulation. 147:66-82
Background: Cardiac hypertrophy increases demands on protein folding, which causes an accumulation of misfolded proteins in the endoplasmic reticulum (ER). These misfolded proteins can be removed by the adaptive retrotranslocation, polyubiquitylation
Autor:
Alina S. Bilal, Sarah N. Parker, Victoria B. Murray, Lauren F. MacDonnell, Donna J. Thuerauf, Christopher C. Glembotski, Erik A. Blackwood
Publikováno v:
Current Protocols. 3
Publikováno v:
Methods in molecular biology (Clifton, N.J.). 2573
Adeno-associated virus serotype 9 (AAV9) is often used in heart research involving gene delivery due to its cardiotropism, high transduction efficiency, and little to no pathogenicity, making it highly applicable for gene manipulation, in vivo. Howev
Publikováno v:
Methods in Molecular Biology ISBN: 9781071627068
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::684fa2a6f1e57758e9d5b0f62394bc85
https://doi.org/10.1007/978-1-0716-2707-5_8
https://doi.org/10.1007/978-1-0716-2707-5_8
Autor:
Donna J. Thuerauf, Cathrine Aivati, Alina S. Bilal, Adrian Arrieta, Christopher C. Glembotski, Michelle Santo Domingo, Shirin Doroudgar, Amber N Pentoney, Winston T Stauffer, Erik A Blackwood, Anup Sarakki
Publikováno v:
The FASEB Journal. 35
We have previously demonstrated that ischemia/reperfusion (I/R) impairs endoplasmic reticulum (ER)-based protein folding in the heart and thereby activates an unfolded protein response sensor and effector, activated transcription factor 6α (ATF6). A
Publikováno v:
J Vis Exp
The isolation and culturing of cardiac myocytes from mice has been essential for furthering the understanding of cardiac physiology and pathophysiology. While isolating myocytes from neonatal mouse hearts is relatively straightforward, myocytes from
Autor:
Anup Sarakki, Cathrine Aivati, Erik A Blackwood, Shirin Doroudgar, Christopher C. Glembotski, Winston T Stauffer, Donna J. Thuerauf, Michelle Santo Domingo, Amber N Pentoney, Adrian Arrieta, Alina S. Bilal
Publikováno v:
J Biol Chem
We have previously demonstrated that ischemia/reperfusion (I/R) impairs endoplasmic reticulum (ER)-based protein folding in the heart and thereby activates an unfolded protein response sensor and effector, activated transcription factor 6α (ATF6). A
Autor:
Adrian Arrieta, Erik A Blackwood, Winston T Stauffer, Alina S. Bilal, Christopher C. Glembotski
Publikováno v:
Cells
Cells, Vol 9, Iss 3, p 602 (2020)
Cells, Vol 9, Iss 3, p 602 (2020)
The heart exhibits incredible plasticity in response to both environmental and genetic alterations that affect workload. Over the course of development, or in response to physiological or pathological stimuli, the heart responds to fluctuations in wo