Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Alexandra M Stoianov"'
Autor:
Alexandra M Stoianov, Debra L Robson, Alexandra M Hetherington, Cynthia G Sawyez, Nica M Borradaile
Publikováno v:
PLoS ONE, Vol 10, Iss 6, p e0131269 (2015)
Elongation factor 1A-1 (eEF1A-1) has non-canonical functions in regulation of the actin cytoskeleton and apoptosis. It was previously identified through a promoter-trap screen as a mediator of fatty acid-induced cell death (lipotoxicity), and was fou
Externí odkaz:
https://doaj.org/article/bcb5f74d543d4e1db84f95f56925ccc0
Autor:
Nica M. Borradaile, Cynthia G. Sawyez, Alexandra M. Hetherington, Emma Zilberman, Debra L. Robson, Alexandra M. Stoianov
Publikováno v:
Cellular Physiology and Biochemistry, Vol 39, Iss 4, Pp 1648-1662 (2016)
Background/Aims: Nonalcoholic fatty liver disease (NAFLD) progression to fibrosis, cirrhosis and hepatocellular carcinoma, alters the cellular composition of this organ. During late-stage NAFLD, fibrotic and possibly cancerous cells can proliferate a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::56bf6cd64aa1ac0f36bb6e41336f18c8
http://www.karger.com/Article/FullText/447866
http://www.karger.com/Article/FullText/447866
Autor:
Debra L. Robson, Nica M. Borradaile, Alexandra M. Stoianov, Cynthia G. Sawyez, Alexandra M. Hetherington
Publikováno v:
PLoS ONE
PLoS ONE, Vol 10, Iss 6, p e0131269 (2015)
PLoS ONE, Vol 10, Iss 6, p e0131269 (2015)
Elongation factor 1A-1 (eEF1A-1) has non-canonical functions in regulation of the actin cytoskeleton and apoptosis. It was previously identified through a promoter-trap screen as a mediator of fatty acid-induced cell death (lipotoxicity), and was fou
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1f4ac1e0ac9259caf6e82c2872d14b1d
https://pubmed.ncbi.nlm.nih.gov/26102086
https://pubmed.ncbi.nlm.nih.gov/26102086
Autor:
Alexandra M. Hetherington, Cynthia G. Sawyez, Emma Zilberman, Alexandra M. Stoianov, Debra L. Robson, Nica M. Borradaile
Publikováno v:
Cellular Physiology and Biochemistry, Vol 39, Iss 4, Pp 1648-1662 (2016)
Background/Aims: Nonalcoholic fatty liver disease (NAFLD) progression to fibrosis, cirrhosis and hepatocellular carcinoma, alters the cellular composition of this organ. During late-stage NAFLD, fibrotic and possibly cancerous cells can proliferate a
Externí odkaz:
https://doaj.org/article/b1356ecf499f4b52921152aac4bb28de