Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Alexander J. Repine"'
Autor:
David M. Guidot, Alexander J. Repine, Stuart L. Bursten, Jack W. Singer, Robert B. Chaney, Glenn C. Rice, Brooks M. Hybertson, John E. Repine
Publikováno v:
American Journal of Physiology-Lung Cellular and Molecular Physiology. 273:L957-L966
We determined that lisofylline, a potent inhibitor of oleate- and linoleate-containing phosphatidic acid formation (half-maximal inhibitory concentration = 40 nM), prevented oxidant-mediated capillary leak in isolated rat lungs given interleukin-8 (I
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::155fb9452877b87cda5b8fe4163e6c0a
https://doi.org/10.1152/ajplung.1997.273.5.l957
https://doi.org/10.1152/ajplung.1997.273.5.l957
Publikováno v:
Inflammation. 21:27-34
Because neutrophils contribute to reperfusion injury associated with acute myocardial infarction (MI), and because tissue plasminogen activator (tPA) is often used in the management of MI, we evaluated the effect of tPA on superoxide (O2.-) productio
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::380d38511d6acb1310943930fe651826
https://doi.org/10.1023/a:1027334607606
https://doi.org/10.1023/a:1027334607606
Autor:
Brooks M. Hybertson, Lance S. Terada, Alexander J. Repine, John E. Repine, Okyong J. Cho, Young Mook Lee, Hyun Gug Cho
Publikováno v:
American journal of physiology. Lung cellular and molecular physiology. 279(1)
Lung lavage fluid of patients with acute lung injury (ALI) has increased levels of interleukin-1 (IL-1) and neutrophils, but their relationship to the lung leak that characterizes these patients is unclear. To address this concern, we investigated th
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b14af759dff7d50214f706fcdd4e25bf
https://pubmed.ncbi.nlm.nih.gov/10893205
https://pubmed.ncbi.nlm.nih.gov/10893205
Autor:
John E. Repine, Brooks M. Hybertson, Lance S. Terada, Hyun Gug Cho, Young Mook Lee, Alexander J. Repine
Publikováno v:
American journal of respiratory and critical care medicine. 155(5)
We hypothesized that phospholipase A2 (PLA2) metabolites contribute to the acute, neutrophil-dependent, edematous lung leak that develops after administration of interleukin-1 (IL-1) intratracheally to rats and tested this premise by using mepacrine
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::00bd10d792297ce2fd9ea596f5b49749
https://pubmed.ncbi.nlm.nih.gov/9154867
https://pubmed.ncbi.nlm.nih.gov/9154867