Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Alexander E. Brady"'
Publikováno v:
Frontiers in Immunology, Vol 14 (2023)
Bronchopulmonary dysplasia (BPD) is a chronic lung disease in preterm birth survivors characterized by inflammation, impaired alveolarization and dysmorphic vasculature. Activated IL-17A+ lymphocytes are key drivers of inflammation in preterm infants
Externí odkaz:
https://doaj.org/article/f43bc4955ae048afb82a11b0a7f217aa
Autor:
Tracy X. Cui, Alexander E. Brady, Ying-Jian Zhang, Christina T. Fulton, Adam M. Goldsmith, Antonia P. Popova
Publikováno v:
Frontiers in Immunology, Vol 14 (2023)
Premature infants with chronic lung disease, bronchopulmonary dysplasia (BPD), develop recurrent cough and wheezing following respiratory viral infections. The mechanisms driving the chronic respiratory symptoms are ill-defined. We have shown that hy
Externí odkaz:
https://doaj.org/article/95849cacabdf48a1a957f76fefc614fe
Publikováno v:
Frontiers in Immunology, Vol 13 (2022)
Prematurity and bronchopulmonary dysplasia (BPD) increase the risk of asthma later in life. Supplemental oxygen therapy is a risk factor for chronic respiratory symptoms in infants with BPD. Hyperoxia induces cell injury and release of damage-associa
Externí odkaz:
https://doaj.org/article/477cfe2086fa49999dbca545c1cf0f3f
Autor:
Tracy X. Cui, Alexander E. Brady, Christina T. Fulton, Ying-Jian Zhang, Liza M. Rosenbloom, Adam M. Goldsmith, Bethany B. Moore, Antonia P. Popova
Publikováno v:
Frontiers in Immunology, Vol 11 (2020)
The histopathology of bronchopulmonary dysplasia (BPD) includes hypoalveolarization and interstitial thickening due to abnormal myofibroblast accumulation. Chorioamnionitis and sepsis are major risk factors for BPD development. The cellular mechanism
Externí odkaz:
https://doaj.org/article/840275bcb20e4c2ba378e68d09ea93ee
Autor:
Tracy X, Cui, Christina T, Fulton, Alexander E, Brady, Ying-Jian, Zhang, Adam M, Goldsmith, Antonia P, Popova
Publikováno v:
Am J Physiol Lung Cell Mol Physiol
Premature infants, especially those with bronchopulmonary dysplasia (BPD), develop recurrent severe respiratory viral illnesses. We have shown that hyperoxic exposure of immature mice, a model of BPD, increases lung IL-12-producing Clec9a(+) CD103(+)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::121539841d8eda31e7c8ad0bb9225d3d
https://pubmed.ncbi.nlm.nih.gov/33112186
https://pubmed.ncbi.nlm.nih.gov/33112186
Autor:
Tracy X. Cui, Alexander E Brady, Antonia P. Popova, Liza M Rosenbloom, Christina T. Fulton, Adam M. Goldsmith
Publikováno v:
D27. MECHANISTIC INSIGHTS INTO BRONCHOPULMONARY DYSPLASIA.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::9c8081bd8eead59afb13477a2dc26a5c
https://doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6033
https://doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6033
In Hyperoxia-Exposed Immature Mice, Gelsolin Protects from Rhinovirus-Induced Pulmonary Inflammation
Publikováno v:
A17. VIRAL INFECTION IN EARLY CHILDHOOD.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::66edeb6d7f33d245a61a1fa8f4665a01
https://doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a1024
https://doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a1024