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pro vyhledávání: '"Alexander Åkesson"'
Autor:
Alexander Åkesson, James B. Bussel, Myriam Martin, Anna M. Blom, Jenny Klintman, Waleed Ghanima, Eva Zetterberg, Lamya Garabet
Publikováno v:
Platelets, Vol 34, Iss 1 (2023)
Increased platelet destruction is central in the pathogenesis of immune thrombocytopenia. However, impaired platelet production is also relevant and its significance underlies the rationale for treatment with thrombopoietin receptor agonists (TPO-RAs
Externí odkaz:
https://doaj.org/article/4ab880211eae4cfa85a11a3095bac1dc
Autor:
Alexander Åkesson, Marcus Ljungkvist, Myriam Martin, Anna M. Blom, Jenny Klintman, Ulf Schött, Eva Zetterberg, Thomas Kander
Publikováno v:
Platelets, Vol 33, Iss 3, Pp 350-359 (2022)
Platelet transfusion refractoriness is a serious clinical concern that complicates the management of thrombocytopenic patients. Previous studies have suggested a potential role for both complement and platelet activation based on in vitro analyses of
Externí odkaz:
https://doaj.org/article/aeb55bbca87c4b1eb6a104d4e605cfd0
Autor:
Alexander Åkesson, Myriam Martin, Anna M. Blom, Eva Zetterberg, Migle Gabrielaite, Jenny Klintman, Maria Rossing
Publikováno v:
Åkesson, A, Martin, M, Blom, A M, Rossing, M, Gabrielaite, M, Zetterberg, E & Klintman, J 2021, ' Clinical characterization and identification of rare genetic variants in atypical hemolytic uremic syndrome : A Swedish retrospective observational study ', Therapeutic Apheresis and Dialysis, vol. 25, no. 6, pp. 988-1000 . https://doi.org/10.1111/1744-9987.13634
INTRODUCTION: Complement-mediated atypical hemolytic uremic syndrome (aHUS) is an ultra-rare renal disease primarily caused by genetic alterations in complement proteins. The genetic work-up required for confirmation of diagnosis is complicated and n
Autor:
Jenny Klintman, Thomas Kander, Ulf Schött, Marcus Ljungkvist, Anna M. Blom, Alexander Åkesson, Myriam Martin, Eva Zetterberg
Publikováno v:
Platelets. 33(3)
Platelet transfusion refractoriness is a serious clinical concern that complicates the management of thrombocytopenic patients. Previous studies have suggested a potential role for both complement and platelet activation based on in vitro analyses of
Publikováno v:
Nephrology. 22:555-561
Aim: Complement-mediated atypical haemolytic uraemic syndrome (aHUS) is a rare disease with high mortality and morbidity if left untreated. The diagnostic work-up is complicated and the manifestations overlap with other conditions. Therefore, we hypo
Publikováno v:
Therapeutic Apheresis and Dialysis. 21:304-319
Complement-mediated atypical hemolytic uremic syndrome (aHUS) is a rare disease associated with high mortality and morbidity. Renal biopsies often indicate thrombotic microangiopathy (TMA). The condition is caused by an excessive activation of the al
Publikováno v:
Therapeutic apheresis and dialysis : official peer-reviewed journal of the International Society for Apheresis, the Japanese Society for Apheresis, the Japanese Society for Dialysis Therapy. 21(4)
Complement-mediated atypical hemolytic uremic syndrome (aHUS) is a rare disease associated with high mortality and morbidity. Renal biopsies often indicate thrombotic microangiopathy (TMA). The condition is caused by an excessive activation of the al
Publikováno v:
Nephrology (Carlton, Vic.). 22(7)
Complement-mediated atypical haemolytic uraemic syndrome (aHUS) is a rare disease with high mortality and morbidity if left untreated. The diagnostic work-up is complicated and the manifestations overlap with other conditions. Therefore, we hypothesi