Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Alessandro A. Bailetti"'
Autor:
Ann E. Collier, Samantha N. Piekos, Angela Liu, Jillian M. Pattison, Franco Felix, Alessandro A. Bailetti, Egor Sedov, Sadhana Gaddam, Hanson Zhen, Anthony E. Oro
Publikováno v:
iScience, Vol 26, Iss 3, Pp 106125- (2023)
Summary: Ectodermal dysplasias including skin abnormalities and cleft lip/palate result from improper surface ectoderm (SE) patterning. However, the connection between SE gene regulatory networks and disease remains poorly understood. Here, we dissec
Externí odkaz:
https://doaj.org/article/afe45ebf4a7949d48c0c76b1a5a59693
Publikováno v:
G3: Genes, Genomes, Genetics, Vol 7, Iss 8, Pp 2427-2438 (2017)
A gain-of-function mutation in the tyrosine kinase JAK2 (JAK2V617F) causes human myeloproliferative neoplasms (MPNs). These patients present with high numbers of myeloid lineage cells and have numerous complications. Since current MPN therapies are n
Externí odkaz:
https://doaj.org/article/3e8b49aadd684781beed978efafe2100
Autor:
Alessandro A. Bailetti, Lenny J. Negrón-Piñeiro, Vishal Dhruva, Sneh Harsh, Sean Lu, Aisha Bosula, Erika A. Bach
Publikováno v:
Disease Models & Mechanisms, Vol 12, Iss 5 (2019)
Myeloproliferative neoplasms (MPNs) are clonal hematopoietic disorders that cause excessive production of myeloid cells. Most MPN patients have a point mutation in JAK2 (JAK2V617F), which encodes a dominant-active kinase that constitutively triggers
Externí odkaz:
https://doaj.org/article/6431b3b620cb4444a4b18048454a0da6
Publikováno v:
G3: Genes, Genomes, Genetics, Vol 7, Iss 8, Pp 2427-2438 (2017)
A gain-of-function mutation in the tyrosine kinase JAK2 (JAK2V617F) causes human myeloproliferative neoplasms (MPNs). These patients present with high numbers of myeloid lineage cells and have numerous complications. Since current MPN therapies are n
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9dbe4dbd67abd17a3db2db7aacf1bfde
https://doi.org/10.1534/g3.117.044172
https://doi.org/10.1534/g3.117.044172
Autor:
Sneh Harsh, Sean Lu, Vishal Dhruva, Lenny J. Negrón-Piñeiro, Aisha Bosula, Erika A. Bach, Alessandro A. Bailetti
Publikováno v:
Disease Models & Mechanisms.
Myeloproliferative neoplasms (MPNs) are clonal hematopoietic disorders that cause excessive production of myeloid cells. Most MPN patients have a point mutation in JAK2 (JAK2V617F), which encodes a dominant-active kinase that constitutively triggers
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::307a417a2bc994919ee90b0ec82aa67d
https://doi.org/10.1242/dmm.038679
https://doi.org/10.1242/dmm.038679
Autor:
Blackwood CA; Molecular Neuropsychiatry Research Branch, National Institutes of Health/National Institute on Drug Abuse Intramural Research Program, Baltimore, MD, United States.; Department of Biomedical Sciences, Cornell University, Ithaca, NY, United States.; Departments of Neuroscience and Genetics, Albert Einstein College of Medicine, New York, NY, United States., Bailetti A; Department of Biomedical Sciences, Cornell University, Ithaca, NY, United States., Nandi S; Departments of Neuroscience and Genetics, Albert Einstein College of Medicine, New York, NY, United States., Gridley T; Maine Medical Center Research Institute, Scarborough, ME, United States., Hébert JM; Departments of Neuroscience and Genetics, Albert Einstein College of Medicine, New York, NY, United States.
Publikováno v:
Frontiers in cell and developmental biology [Front Cell Dev Biol] 2020 Feb 26; Vol. 8, pp. 113. Date of Electronic Publication: 2020 Feb 26 (Print Publication: 2020).