Zobrazeno 1 - 10
of 24
pro vyhledávání: '"Alessandra Venanzi"'
Autor:
Lorenza Falini, Alessandra Venanzi, Valentina Tini, Alessandra Innocente, Stelvio Ballanti, Simonetta Saldi, Silvio Sivolella, Antonio Pierini, Cynthia Aristei, Enrico Tiacci, Vincenzo Maria Perriello, Brunangelo Falini
Publikováno v:
Haematologica, Vol 108, Iss 1 (2022)
Externí odkaz:
https://doaj.org/article/dcc18fec327f45a78e52dff6f47c3286
Autor:
Oxana Bereshchenko, Maddalena Coppo, Stefano Bruscoli, Michele Biagioli, Monica Cimino, Tiziana Frammartino, Daniele Sorcini, Alessandra Venanzi, Moises Di Sante, Carlo Riccardi
Publikováno v:
Cell Reports, Vol 7, Iss 2, Pp 464-475 (2014)
Summary: Regulatory T (Treg) cells expressing the transcription factor forkhead box P3 (FoxP3) control immune responses and prevent autoimmunity. Treatment with glucocorticoids (GCs) has been shown to increase Treg cell frequency, but the mechanisms
Externí odkaz:
https://doaj.org/article/27a98c24410440bd84dfce7488e17385
Autor:
Enrico Tiacci, Brunangelo Falini, Stefano Ascani, Paolo Sportoletti, Maria Paola Martelli, Sofia Sciabolacci, Lorenza Falini, Alessandra Pucciarini, Luisa Tasselli, Simona Ultimo, Valentina Pettirossi, Alessia Santi, Roberto Limongello, Sara G. Milner, Gianluca Schiavoni, Andrea Marra, Alessandra Venanzi
Supplementary Appendix includes Supplementary Data and Discussion.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::95052e2802621e6d70df34826a1be1b4
https://doi.org/10.1158/2643-3230.22543447.v1
https://doi.org/10.1158/2643-3230.22543447.v1
Autor:
Enrico Tiacci, Brunangelo Falini, Stefano Ascani, Paolo Sportoletti, Maria Paola Martelli, Sofia Sciabolacci, Lorenza Falini, Alessandra Pucciarini, Luisa Tasselli, Simona Ultimo, Valentina Pettirossi, Alessia Santi, Roberto Limongello, Sara G. Milner, Gianluca Schiavoni, Andrea Marra, Alessandra Venanzi
Supplementary Figures show: i) Fig. S1: Microdissection of HRS cells and reactive lymphocytes from the cHL tissue; ii) Fig. S2: Tissue involvement by clonal hematopoiesis (in cHL Case 2; iii) Fig.S3: T-cell receptor gamma gene rearrangement analysis
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::32ed18fc779df40e78eda3ff3221a1d3
https://doi.org/10.1158/2643-3230.22543444.v1
https://doi.org/10.1158/2643-3230.22543444.v1
Autor:
Enrico Tiacci, Brunangelo Falini, Stefano Ascani, Paolo Sportoletti, Maria Paola Martelli, Sofia Sciabolacci, Lorenza Falini, Alessandra Pucciarini, Luisa Tasselli, Simona Ultimo, Valentina Pettirossi, Alessia Santi, Roberto Limongello, Sara G. Milner, Gianluca Schiavoni, Andrea Marra, Alessandra Venanzi
Supplementary Tables show: i) Supplementary Table 1. Genes subjected to targeted deep sequencing; ii) Supplementary Table 2. CHIP-associated mutations observed by NGS in the cohort of 39 cHL patients; iii)Supplementary Table 3. List of somatic mutati
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a6dc0d1ce1322a845f7f253ac3427982
https://doi.org/10.1158/2643-3230.22543441
https://doi.org/10.1158/2643-3230.22543441
Autor:
Enrico Tiacci, Brunangelo Falini, Stefano Ascani, Paolo Sportoletti, Maria Paola Martelli, Sofia Sciabolacci, Lorenza Falini, Alessandra Pucciarini, Luisa Tasselli, Simona Ultimo, Valentina Pettirossi, Alessia Santi, Roberto Limongello, Sara G. Milner, Gianluca Schiavoni, Andrea Marra, Alessandra Venanzi
Clonal hematopoiesis predisposes to hematologic malignancies. However, clonal hematopoiesis is understudied in classic Hodgkin lymphoma (cHL), a mature B-cell neoplasm exhibiting the most abundant microenvironment. We analyzed clonal hematopoiesis in
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::713d8accdbacb0a7dde465d90f773f9d
https://doi.org/10.1158/2643-3230.c.6550264
https://doi.org/10.1158/2643-3230.c.6550264
Autor:
Valentina Pettirossi, Alessandra Venanzi, Ariele Spanhol-Rosseto, Gianluca Schiavoni, Alessia Santi, Luisa Tasselli, Marta Naccari, Valentina Pensato, Alessandra Pucciarini, Maria Paola Martelli, Hans Drexler, Brunangelo Falini, Enrico Tiacci
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c39f3d460fd79122c41ed9630952d0d0
https://hdl.handle.net/11391/1544253
https://hdl.handle.net/11391/1544253
Autor:
Jad Othman, Manja Meggendorfer, Enrico Tiacci, Christian Thiede, Richard Schlenk, Richard Dillon, Sebastian Stasik, Alessandra Venanzi, Sarah Bertoli, Eric Delabesse, Pierre-Yves Dumas, Arnaud Pigneux, Audrey Bidet, Amanda F. Gilkes, Ian Thomas, Maria Teresa Voso, Alessandro Rambaldi, Lorenzo Brunetti, Vincenzo M. Perriello, Vibeke Andresen, Bjorn T. Gjertsen, Maria Paola Martelli, Christian Récher, Christoph Röllig, Martin Bornhäuser, Hubert Serve, Carsten Müller-Tidow, Claudia D. Baldus, Tortsten Haferlach, Nigel Russell, Brunangelo Falini
Publikováno v:
Blood
NPM 1-mutated acute myeloid leukemia (AML) shows unique features. However, the characteristics of “therapy-related” NPM1-mutated AML (t-NPM1 AML) are poorly understood. We compared the genetics, transcriptional profile, and clinical outcomes of t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::04aec7fe5258ce236c8f2fd58ecec55b
Autor:
Giovanni Martino, Roberta Rossi, Maria Paola Martelli, Alessandra Venanzi, Paolo Sportoletti, Enrico Tiacci, Giuseppe Avvisati, Ombretta Annibali, Maria Grazia Mameli, Brunangelo Falini
Publikováno v:
Genes, Vol 12, Iss 1426, p 1426 (2021)
Genes
Volume 12
Issue 9
Genes
Volume 12
Issue 9
Nucleophosmin (NPM1) mutations occurring in acute myeloid leukemia (AML) (about 50 so far identified) cluster almost exclusively in exon 12 and lead to common changes at the NPM1 mutants C-terminus, i.e., loss of tryptophans 288 and 290 (or 290 alone
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::45f0c582b20db97d61f4c9c29a0efc32
https://www.mdpi.com/2073-4425/12/9/1426
https://www.mdpi.com/2073-4425/12/9/1426
Autor:
Alessandra, Venanzi, Roberta, Rossi, Giovanni, Martino, Ombretta, Annibali, Giuseppe, Avvisati, Maria Grazia, Mameli, Paolo, Sportoletti, Enrico, Tiacci, Brunangelo, Falini, Maria Paola, Martelli
Publikováno v:
Genes
Nucleophosmin (NPM1) mutations occurring in acute myeloid leukemia (AML) (about 50 so far identified) cluster almost exclusively in exon 12 and lead to common changes at the NPM1 mutants C-terminus, i.e., loss of tryptophans 288 and 290 (or 290 alone
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::82757fa025ee7f3f7c7660ffbf264faf
https://pubmed.ncbi.nlm.nih.gov/34573408
https://pubmed.ncbi.nlm.nih.gov/34573408