Zobrazeno 1 - 2
of 2
pro vyhledávání: '"Alessandra Francone"'
Autor:
Annalise Di Marco, Alberto Bresciani, Simona Ponzi, Antonella Cellucci, Jesus Maria Ontoria Ontoria, Alina Ciammaichella, Alessandra Francone, Steven J. Harper, Savina Malancona, Maria Veneziano, Vincenzo Summa, Edith Monteagudo, Federica Ferrigno, Emanuela Nizi, Ilaria Biancofiore, Ilaria Rossetti, Paola Pace
Publikováno v:
ACS Medicinal Chemistry Letters. 10:481-486
[Image: see text] The application of class I HDAC inhibitors as cancer therapies is well established, but more recently their development for nononcological indications has increased. We report here on the generation of improved class I selective hum
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::02cfa3c775b7676f93176df62c7a4015
https://doi.org/10.1021/acsmedchemlett.8b00517
https://doi.org/10.1021/acsmedchemlett.8b00517
Autor:
Sergio Altamura, Antonio Baldini, Monica Bisbocci, Gabriella Lania, Vincenza Colonna, Alberto Bresciani, Alessandra Francone
Publikováno v:
Scopus-Elsevier
Human Molecular Genetics
Human molecular genetics
(2017). doi:10.1093/hmg/ddx353
info:cnr-pdr/source/autori:Lania G, Bresciani A, Bisbocci M, Francone A, Colonna V, Altamura S, Baldini A./titolo:Vitamin B12 ameliorates the phenotype of a mouse model of DiGeorge syndrome./doi:10.1093%2Fhmg%2Fddx353/rivista:Human molecular genetics (Print)/anno:2017/pagina_da:/pagina_a:/intervallo_pagine:/volume
25 (2016): 4369–4375. doi:10.1093/hmg/ddw267
info:cnr-pdr/source/autori:Lania G, Bresciani A, Bisbocci M, Francone A, Colonna V, Altamura S, Baldini A./titolo:Vitamin B12 ameliorates the phenotype of a mouse model of DiGeorge syndrome./doi:10.1093%2Fhmg%2Fddw267/rivista:Human molecular genetics (Print)/anno:2016/pagina_da:4369/pagina_a:4375/intervallo_pagine:4369–4375/volume:25
Human Molecular Genetics
Human molecular genetics
(2017). doi:10.1093/hmg/ddx353
info:cnr-pdr/source/autori:Lania G, Bresciani A, Bisbocci M, Francone A, Colonna V, Altamura S, Baldini A./titolo:Vitamin B12 ameliorates the phenotype of a mouse model of DiGeorge syndrome./doi:10.1093%2Fhmg%2Fddx353/rivista:Human molecular genetics (Print)/anno:2017/pagina_da:/pagina_a:/intervallo_pagine:/volume
25 (2016): 4369–4375. doi:10.1093/hmg/ddw267
info:cnr-pdr/source/autori:Lania G, Bresciani A, Bisbocci M, Francone A, Colonna V, Altamura S, Baldini A./titolo:Vitamin B12 ameliorates the phenotype of a mouse model of DiGeorge syndrome./doi:10.1093%2Fhmg%2Fddw267/rivista:Human molecular genetics (Print)/anno:2016/pagina_da:4369/pagina_a:4375/intervallo_pagine:4369–4375/volume:25
Pathological conditions caused by reduced dosage of a gene, such as gene haploinsufficiency, can potentially be reverted by enhancing the expression of the functional allele. In practice, low specificity of therapeutic agents, or their toxicity reduc
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::65403d77703fedaac64046b8d33dc720
http://hdl.handle.net/11588/643539
http://hdl.handle.net/11588/643539
