Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Aleksandar Atemin"'
Autor:
Georgi Danovski, Greta Panova, Bradley Keister, Georgi Georgiev, Aleksandar Atemin, Sonya Uzunova, Rumen Stamatov, Petar-Bogomil Kanev, Radoslav Aleksandrov, Krastan B. Blagoev, Stoyno S. Stoynov
Publikováno v:
iScience, Vol 27, Iss 9, Pp 110826- (2024)
Summary: During DNA repair, ATM-induced H2AX histone phosphorylation and MDC1 recruitment spread megabases beyond the damage site. While loop extrusion has been suggested to drive this spread, the underlying mechanism remains unclear. Herein, we prov
Externí odkaz:
https://doaj.org/article/14a8058a4f0c4a75968a8af700c246cf
Autor:
Aleksandar Atemin, Aneliya Ivanova, Wiley Peppel, Rumen Stamatov, Rodrigo Gallegos, Haley Durden, Sonya Uzunova, Michael D. Vershinin, Saveez Saffarian, Stoyno S. Stoynov
Publikováno v:
Viruses, Vol 16, Iss 8, p 1341 (2024)
The efficiency of virus internalization into target cells is a major determinant of infectivity. SARS-CoV-2 internalization occurs via S-protein-mediated cell binding followed either by direct fusion with the plasma membrane or endocytosis and subseq
Externí odkaz:
https://doaj.org/article/4831920d927c41f298cc638c4553658b
Autor:
Aleksandar Atemin, Aneliya Ivanova, Petar-Bogomil Kanev, Sonya Uzunova, Marina Nedelcheva-Veleva, Stoyno Stoynov
Publikováno v:
International Journal of Molecular Sciences, Vol 25, Iss 15, p 8230 (2024)
The measurement of dynamic changes in protein level and localization throughout the cell cycle is of major relevance to studies of cellular processes tightly coordinated with the cycle, such as replication, transcription, DNA repair, and checkpoint c
Externí odkaz:
https://doaj.org/article/14114fdc32b84f8781206016431d0c27
Autor:
Aneliya Ivanova, Aleksandar Atemin, Sonya Uzunova, Georgi Danovski, Radoslav Aleksandrov, Stoyno Stoynov, Marina Nedelcheva-Veleva
Publikováno v:
Molecules, Vol 27, Iss 1, p 97 (2021)
Cells have evolved elaborate mechanisms to regulate DNA replication machinery and cell cycles in response to DNA damage and replication stress in order to prevent genomic instability and cancer. The E3 ubiquitin ligase SCFDia2 in S. cerevisiae is inv
Externí odkaz:
https://doaj.org/article/47e1c3ad3df64f63b6836000aa4ab9b0
Publikováno v:
Biomedicines, Vol 9, Iss 9, p 1238 (2021)
Cells are constantly exposed to numerous mutagens that produce diverse types of DNA lesions. Eukaryotic cells have evolved an impressive array of DNA repair mechanisms that are able to detect and repair these lesions, thus preventing genomic instabil
Externí odkaz:
https://doaj.org/article/973f230d8c034e70841e5d6fde147eae
Publikováno v:
Biomedicines, Vol 9, Iss 1238, p 1238 (2021)
Biomedicines; Volume 9; Issue 9; Pages: 1238
Biomedicines
Biomedicines; Volume 9; Issue 9; Pages: 1238
Biomedicines
Cells are constantly exposed to numerous mutagens that produce diverse types of DNA lesions. Eukaryotic cells have evolved an impressive array of DNA repair mechanisms that are able to detect and repair these lesions, thus preventing genomic instabil
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::09fd93bdf382af99bbef8f3668bdb84c
https://www.mdpi.com/2227-9059/9/9/1238
https://www.mdpi.com/2227-9059/9/9/1238
Autor:
Stoyno S. Stoynov, Anthony A. Hyman, Radoslav Aleksandrov, Mihail Sarov, Georgi Danovski, Greta Panova, Teodora Dyankova, Stephan W. Grill, Ina Poser, Anton Dotchev, Yordan Babukov, Aneliya Ivanova, Georgi P. Georgiev, Aleksandar Atemin, Lars Hubatsch, Frank Buchholz, Dragomir B. Krastev, Susanne Hasse, Marina N. Nedelcheva-Veleva
Publikováno v:
Molecular cell. 69(6)
A single mutagen can generate multiple different types of DNA lesions. How different repair pathways cooperate in complex DNA lesions, however, remains largely unclear. Here we measured, clustered, and modeled the kinetics of recruitment and dissocia
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1207938fca0ff4448138af7c15eba821
https://pubmed.ncbi.nlm.nih.gov/29547717
https://pubmed.ncbi.nlm.nih.gov/29547717