Zobrazeno 1 - 10
of 16
pro vyhledávání: '"Alejandro Galindo-Tovar"'
Evolution of the consumption of lorazepam in a rural environment in the Region of Murcia (2012-2018)
Publikováno v:
Pharmaceutical Care España, Vol 24, Iss 4 (2022)
Objective: To evaluate the consumption of lorazepam in the Region of Murcia, specifically in the district of Mula, during the period 2012-2018, considering age and sex variables. Method: Cross-sectional descriptive study, where the use of benzodiaz
Externí odkaz:
https://doaj.org/article/ee2d861e43a94bcd82fa0c4cd3631d10
Autor:
Francisco Valdivia-Muñoz, Jorge Simal-Aguado, Alejandro Galindo-Tovar, Juan-Antonio García-Carmona, María Pilar Campos-Navarro
Publikováno v:
International Clinical Psychopharmacology. 36:18-24
To date, only a few studies compared some long-acting injectables (LAIs) antipsychotics showing similar symptom improvement, relapse rates and adherence to treatment. We evaluated the use of LAIs antipsychotics [aripiprazole-1-month (A1M); paliperido
Autor:
Jorge Simal-Aguado, María Pilar Campos-Navarro, Juan-Antonio García-Carmona, Francisco Valdivia-Muñoz, Alejandro Galindo-Tovar
Publikováno v:
Clinical Drug Investigation. 40:459-468
Long-acting injectable antipsychotics (LAIs) have been widely studied in schizophrenia and evidence suggests that they could be also used for the treatment of bipolar and schizoaffective disorders. Nonetheless, there are no studies evaluating their r
Autor:
Francisco Valdivia-Muñoz, María Pilar Campos-Navarro, Juan-Antonio García-Carmona, Alejandro Galindo-Tovar, Jorge Simal-Aguado
Publikováno v:
International clinical psychopharmacology. 36(4)
The aim of the present study was to evaluate the use of oral vs. long-acting injectables (LAIs) antipsychotics, as well as, to compare the effectiveness of different LAI antipsychotics [aripiprazole-1-month, paliperidone-1-month (PP1M), paliperidone-
Publikováno v:
Naunyn-Schmiedeberg's Archives of Pharmacology. 389:177-186
Phosphodiesterases PDE2, PDE3, and PDE4 are expressed in murine sinoatrial cells. PDE3 and/or PDE4 reduce heart rate but apparently do not influence the tachycardia mediated through sinoatrial β1- and β2-adrenoceptors despite the high content of si
Autor:
Alejandro Galindo-Tovar, R.I. Hussain, Peter C. M. Molenaar, Kurt A. Krobert, Torsten Christ, Katherine T. Gillette, Finn Olav Levy, Lu Chen, Emanuel Berk, Alberto J. Kaumann, Andreas Engel, Ursula Ravens
Publikováno v:
British Journal of Pharmacology. 169:528-538
Background and purpose Phosphodiesterases PDE3 and/or PDE4 control ventricular effects of catecholamines in several species but their relative effects in failing human ventricle are unknown. We investigated whether the PDE3-selective inhibitor cilost
Publikováno v:
Naunyn-Schmiedeberg's Archives of Pharmacology. 379:379-384
Acting through a low-affinity site of the beta(1)-adrenoceptor (beta(1L)AR), CGP12177 causes sinoatrial tachycardia and positive inotropic effects in left atrium but not in the ventricle of the rat. However, inhibition of either PDE3 or PDE4 also unc
Publikováno v:
British Journal of Pharmacology. 153:710-720
Background and purpose: β1 and β2-adrenoceptors coexist in murine heart but β2-adrenoceptor-mediated effects have not been detected in atrial and ventricular tissues, possibly due to marked phosphodiesterase (PDE) activity. We investigated the inf
Publikováno v:
Naunyn-Schmiedeberg's archives of pharmacology. 389(2)
Phosphodiesterases PDE2, PDE3, and PDE4 are expressed in murine sinoatrial cells. PDE3 and/or PDE4 reduce heart rate but apparently do not influence the tachycardia mediated through sinoatrial β1- and β2-adrenoceptors despite the high content of si
Autor:
Torsten Christ, Alberto J. Kaumann, Andreas Engel, Emanuel Berk, Peter C. M. Molenaar, Katherine T. Gillette, Ursula Ravens, Alejandro Galindo-Tovar
Publikováno v:
Naunyn-Schmiedeberg's archives of pharmacology. 387(7)
The beta-blockers carvedilol and metoprolol provide important therapeutic strategies for heart failure treatment. Therapy with metoprolol facilitates the control by phosphodiesterase PDE3, but not PDE4, of inotropic effects of catecholamines in human