Výsledky vyhledávání - "Alejandro D Nadra"

Akademický článek

Transcription factor specificity limits the number of DNA-binding motifs.

Autor: Ariel A Aptekmann, Denys Bulavka, Alejandro D Nadra, Ignacio E Sánchez

Publikováno v: PLoS ONE, Vol 17, Iss 1, p e0263307 (2022)

We study the limits imposed by transcription factor specificity on the maximum number of binding motifs that can coexist in a gene regulatory network, using the SwissRegulon Fantom5 collection of 684 human transcription factor binding sites as a mode

Externí odkaz: https://doaj.org/article/f91d83dcecd84016a4529356a9ba69fb

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Akademický článek

Circadian period integrates network information through activation of the BMP signaling pathway.

Autor: Esteban J Beckwith, E Axel Gorostiza, Jimena Berni, Carolina Rezával, Agustín Pérez-Santángelo, Alejandro D Nadra, María Fernanda Ceriani

Publikováno v: PLoS Biology, Vol 11, Iss 12, p e1001733 (2013)

Living organisms use biological clocks to maintain their internal temporal order and anticipate daily environmental changes. In Drosophila, circadian regulation of locomotor behavior is controlled by ∼150 neurons; among them, neurons expressing the

Externí odkaz: https://doaj.org/article/184328e1e7ad45ac8711e332b857ec4f

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Akademický článek

Biophysical characterisation of neuroglobin of the icefish, a natural knockout for hemoglobin and myoglobin. Comparison with human neuroglobin.

Autor: Daniela Giordano, Ignacio Boron, Stefania Abbruzzetti, Wendy Van Leuven, Francesco P Nicoletti, Flavio Forti, Stefano Bruno, C-H Christina Cheng, Luc Moens, Guido di Prisco, Alejandro D Nadra, Darío Estrin, Giulietta Smulevich, Sylvia Dewilde, Cristiano Viappiani, Cinzia Verde

Publikováno v: PLoS ONE, Vol 7, Iss 12, p e44508 (2012)

The Antarctic icefish Chaenocephalus aceratus lacks the globins common to most vertebrates, hemoglobin and myoglobin, but has retained neuroglobin in the brain. This conserved globin has been cloned, over-expressed and purified. To highlight similari

Externí odkaz: https://doaj.org/article/b9f7268e7c4140a3b2f62ffbeed68267

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Akademický článek

Structure-based analysis of five novel disease-causing mutations in 21-hydroxylase-deficient patients.

Autor: Carolina Minutolo, Alejandro D Nadra, Cecilia Fernández, Melisa Taboas, Noemí Buzzalino, Bárbara Casali, Susana Belli, Eduardo H Charreau, Liliana Alba, Liliana Dain

Publikováno v: PLoS ONE, Vol 6, Iss 1, p e15899 (2011)

Congenital adrenal hyperplasia (CAH) due to 21-hydroxylase deficiency is the most frequent inborn error of metabolism, and accounts for 90-95% of CAH cases. The affected enzyme, P450C21, is encoded by the CYP21A2 gene, located together with a 98% nuc

Externí odkaz: https://doaj.org/article/0b3ff7a736854c4088db0fd43b6b4937

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