Zobrazeno 1 - 10
of 59
pro vyhledávání: '"Alda Tufro"'
Publikováno v:
Frontiers in Pharmacology, Vol 12 (2022)
Vascular endothelial growth factor-a (VEGF-A) and nitric oxide (NO) are essential for glomerular filtration barrier homeostasis, and are dysregulated in diabetic kidney disease (DKD). While NO availability is consistently low in diabetes, both high a
Externí odkaz:
https://doaj.org/article/41ddbebcb869477b9a37fab161a12f88
Publikováno v:
Frontiers in Medicine, Vol 8 (2021)
The molecular pathogenesis of diabetic kidney disease progression is complex and remains unresolved. Rho-GAP MYO9A was recently identified as a novel podocyte protein and a candidate gene for monogenic FSGS. Myo9A involvement in diabetic kidney disea
Externí odkaz:
https://doaj.org/article/d184930072104e18864639542ac0344e
Autor:
Heon Yung Gee, Carolin E. Sadowski, Pardeep K. Aggarwal, Jonathan D. Porath, Toma A. Yakulov, Markus Schueler, Svjetlana Lovric, Shazia Ashraf, Daniela A. Braun, Jan Halbritter, Humphrey Fang, Rannar Airik, Virginia Vega-Warner, Kyeong Jee Cho, Timothy A. Chan, Luc G. T. Morris, Charles ffrench-Constant, Nicholas Allen, Helen McNeill, Rainer Büscher, Henriette Kyrieleis, Michael Wallot, Ariana Gaspert, Thomas Kistler, David V. Milford, Moin A. Saleem, Wee Teik Keng, Stephen I. Alexander, Rudolph P. Valentini, Christoph Licht, Jun C. Teh, Radovan Bogdanovic, Ania Koziell, Agnieszka Bierzynska, Neveen A. Soliman, Edgar A. Otto, Richard P. Lifton, Lawrence B. Holzman, Nicholas E. S. Sibinga, Gerd Walz, Alda Tufro, Friedhelm Hildebrandt
Publikováno v:
Nature Communications, Vol 7, Iss 1, Pp 1-11 (2016)
Steroid-sensitive nephrotic syndrome (SRNS) can cause CKD and necessitate kidney transplant. Here the authors identify FAT1 mutations by homozygosity mapping and whole-exome sequencing in families with SRNS and provide functional mouse and zebrafish
Externí odkaz:
https://doaj.org/article/28d36207928640028a9a892d5c32fd4f
Autor:
Katherine Carranza, Dolores Veron, Alicia Cercado, Noemi Bautista, Wilson Pozo, Alda Tufro, Delma Veron
Publikováno v:
Nefrología (English Edition), Vol 35, Iss 2, Pp 131-138 (2015)
The prevalence of diabetes mellitus increased during the last century and it is estimated that 45% of the patients are not diagnosed. In South America the prevalence of diabetes and chronic kidney disease (CKD) increased, with a great disparity among
Externí odkaz:
https://doaj.org/article/a833e94225044a5abda0a271d1334ef0
Autor:
Katherine Carranza, Dolores Veron, Alicia Cercado, Noemi Bautista, Wilson Pozo, Alda Tufro, Delma Veron
Publikováno v:
Nefrología, Vol 35, Iss 2, Pp 131-138 (2015)
La prevalencia de diabetes mellitus aumentó en el último siglo y se estima que el 45% de los pacientes, no estarían diagnosticados. En Sudamérica la prevalencia de diabetes y de enfermedad renal crónica (ERC) incrementó, existiendo gran dispari
Externí odkaz:
https://doaj.org/article/81b0f4820c79430a92b5c3cba44751b1
Autor:
Delma Veron, Alda Tufro
Publikováno v:
Nefrología, Vol 36, Iss 6, Pp 706-707 (2016)
Externí odkaz:
https://doaj.org/article/8cb9f4f8e46f4df48d145ae6cad5b38e
Autor:
Delma Veron, Alda Tufro
Publikováno v:
Nefrología (English Edition), Vol 36, Iss 6, Pp 706-707 (2016)
Externí odkaz:
https://doaj.org/article/25ece34eda064dd7a995b1d8ed926ec6
Autor:
Delma Veron, Guillermo Villegas, Pardeep Kumar Aggarwal, Claudia Bertuccio, Juan Jimenez, Heino Velazquez, Kimberly Reidy, Dale R Abrahamson, Gilbert Moeckel, Michael Kashgarian, Alda Tufro
Publikováno v:
PLoS ONE, Vol 7, Iss 7, p e40589 (2012)
Podocyte or endothelial cell VEGF-A knockout causes thrombotic microangiopathy in adult mice. To study the mechanism involved in acute and local injury caused by low podocyte VEGF-A we developed an inducible, podocyte-specific VEGF-A knockdown mouse,
Externí odkaz:
https://doaj.org/article/e158b1c784ad483b8ca2e989cd6b48dd
Autor:
Delma, Veron, Pardeep K, Aggarwal, Qi, Li, Gilbert, Moeckel, Michael, Kashgarian, Alda, Tufro
Publikováno v:
Frontiers in pharmacology. 12
Vascular endothelial growth factor-a (VEGF-A) and nitric oxide (NO) are essential for glomerular filtration barrier homeostasis, and are dysregulated in diabetic kidney disease (DKD). While NO availability is consistently low in diabetes, both high a
Publikováno v:
Pediatric Nephrology (Berlin, Germany)
The synchronized advent of high-throughput next-generation sequencing technology and knowledge of the human genome has rendered exponential contributions to our understanding of the pathophysiology of glomerular kidney diseases. A genetic diagnosis c