Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Akos Hegyi"'
Autor:
Akos Hegyi, Oktavia Tarjanyi, Tibor T. Glant, Katalin Mikecz, Tibor A. Rauch, Ferenc Boldizsár, Katalin Olasz
Publikováno v:
Cardiovascular Pathology. 25:353-361
Although dilated cardiomyopathy (DCM) is often caused by viral infections, it frequently involves autoimmune mechanisms associated with particular HLA-DR and DQ alleles. Our homozygous HLA-DQ8Ab(0) transgenic mice in the BALB/c background (HLA-DQ8(BA
Autor:
Katalin Olasz, Anna Laszlo, Katalin Mikecz, Ferenc Boldizsár, Akos Hegyi, Alison Finnegan, Gyorgy Nagyeri, Tibor T. Glant, Marianna Radacs
Publikováno v:
Arthritis & Rheumatism. 63:1312-1321
Objective To develop a simplified and relatively inexpensive version of cartilage proteoglycan–induced arthritis (PGIA), an autoimmunity model of rheumatoid arthritis (RA), and to evaluate the extent to which this new model replicates the disease p
Autor:
Katalin Olasz, Akos Hegyi, W. van Eden, Tibor T. Glant, Katalin Mikecz, Ferenc Boldizsár, Tibor A. Rauch, Katalin Kis-Toth, Oktavia Tarjanyi
Publikováno v:
Clinical and experimental immunology. 167(2)
Summary T cell receptor transgenic (TCR-Tg) mice specific for the arthritogenic 5/4E8 epitope in the G1 domain of cartilage proteoglycan were generated and back-crossed into arthritis-prone BALB/c background. Although more than 90% of CD4+ T cells of
Autor:
Katalin Olasz, Akos Hegyi, Katalin Mikecz, Oktavia Tarjanyi, Katalin Kis-Toth, Tibor T. Glant, Ferenc Boldizsár
Publikováno v:
Arthritis and rheumatism. 62(10)
Objective To investigate whether genetic preponderance of a T cell receptor (TCR) recognizing an arthritogenic peptide of human cartilage proteoglycan (PG) is sufficient for development of arthritis. Methods We performed a longitudinal study using BA