Zobrazeno 1 - 10
of 142
pro vyhledávání: '"Akira ISHISAKI"'
Autor:
Naoyuki Chosa, Akira Ishisaki
Publikováno v:
Japanese Dental Science Review, Vol 54, Iss 1, Pp 37-44 (2018)
Summary: Mesenchymal stem cells (MSCs) retain the ability to self-renew and differentiate into mesenchymal cells. Therefore, human MSCs are suitable candidates for use in regenerative medicine and cell therapies. Upon activation by tissue damage, MSC
Externí odkaz:
https://doaj.org/article/4547b0f3ca1b4e1f87a56430a71f8ac2
Autor:
Sachiko KURAMITSU-FUJIMOTO, Wataru ARIYOSHI, Noriko SAITO, Toshinori OKINAGA, Masaharu KAMO, Akira ISHISAKI, Takashi TAKATA, Kazunori YAMAGUCHI, Tatsuji NISHIHARA
Publikováno v:
Journal of Applied Oral Science, Vol 23, Iss 1, Pp 49-55 (2015)
Objective Enamel matrix derivative (EMD) is used clinically to promote periodontal tissue regeneration. However, the effects of EMD on gingival epithelial cells during regeneration of periodontal tissues are unclear. In this in vitro study, we purifi
Externí odkaz:
https://doaj.org/article/f72735304e33468481b904ddb50cb259
Autor:
Keita Suzuki, Naoyuki Chosa, Shunsuke Sawada, Naoki Takizawa, Takashi Yaegashi, Akira Ishisaki
Publikováno v:
Stem Cells International, Vol 2017 (2017)
Mesenchymal stem cells (MSCs) are involved in anti-inflammatory events and tissue repair; these functions are activated by their migration or homing to inflammatory tissues in response to various chemokines. However, the mechanism by which MSCs inter
Externí odkaz:
https://doaj.org/article/abb94e5e476f48cfab1e2ddb67130d52
Autor:
Hitomichi Kimura, Naoto Okubo, Naoyuki Chosa, Seiko Kyakumoto, Masaharu Kamo, Hiroyuki Miura, Akira Ishisaki
Publikováno v:
Cellular Physiology and Biochemistry, Vol 32, Iss 4, Pp 899-914 (2013)
Background/Aims: Remodeling of fibrous and vascular tissues in the periodontal ligament (PDL) around the tooth root was observed during tooth movement by orthodontic force application. We previously demonstrated that a single cell-derived culture (SC
Externí odkaz:
https://doaj.org/article/f9d5d5e9f1ac4d7f9dcb3db19796a37f
Autor:
Kanna Asanuma, Seiji Yokota, Naoyuki Chosa, Masaharu Kamo, Miho Ibi, Hisayo Mayama, Tarou Irié, Kazuro Satoh, Akira Ishisaki
Publikováno v:
Journal of oral biosciences.
Temporomandibular joint osteoarthritis (TMJ-OA) is a multifactorial disease caused by inflammation and oxidative stress. It has been hypothesized that mechanical stress-induced injury of TMJ tissues induces the generation of reactive oxygen species (
Publikováno v:
Molecular biology reports.
Temporomandibular joint osteoarthritis (TMJ-OA) causes cartilage degeneration, bone cavitation, and fibrosis of the TMJ. However, the mechanisms underlying the fibroblast-like synoviocyte (FLS)-mediated inflammatory activity in TMJ-OA remain unclear.
Autor:
Haruka Kaneko, Shinichiro Kuroshima, Takashi Sawase, Ryohei Kozutsumi, Muneteru Sasaki, Akira Ishisaki
Publikováno v:
Calcified tissue international. 110(1)
The pathophysiology, histopathology, and immunopathology of bisphosphonate-related osteonecrosis of the jaw (BRONJ) Stage 0 remain unclear. The aim of this study was to investigate the effects of high-dose bisphosphonates on tooth extraction socket h
Autor:
Masahito Ogasawara, Hisayo Fukuda, Taichi Ishikawa, Keizo Tokuhiro, Masaharu Kamo, Hiroyuki Yamada, Akira Ishisaki
Publikováno v:
Proceedings for Annual Meeting of The Japanese Pharmacological Society. 96:2-B
Autor:
Saki Tamaki, Takashi Sawase, Kazunori Nakajima, Akira Ishisaki, Shinichiro Kuroshima, Hiroe Kakehashi, Hiroki Hayano
Publikováno v:
Bone. 141
Denosumab-related osteonecrosis of the jaw (DRONJ), which mainly occurs in cancer patients receiving anti-receptor activator NF-kappaB ligand (RANKL) antibody, reduces oral health-related quality of life. However, the exact mechanisms of and definiti