Zobrazeno 1 - 10
of 12
pro vyhledávání: '"Ajinkya, Kumbhare"'
Publikováno v:
Frontiers in Immunology, Vol 8 (2017)
Myeloid precursor cell reprogramming into a myeloid-derived suppressor cell (MDSC) contributes to high mortality rates in mouse and human sepsis. S100A9 mRNA and intracellular protein levels increase during early sepsis and remain elevated in Gr1+CD1
Externí odkaz:
https://doaj.org/article/3c9da835a62040f5a41bdacc3decc440
Publikováno v:
Molecular Immunology. 150:246
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::599e186f1b3fbe72a7035a2854d6cd3c
https://doi.org/10.1016/j.molimm.2022.06.004
https://doi.org/10.1016/j.molimm.2022.06.004
Autor:
Tuqa, Alkhateeb, Isatou, Bah, Ajinkya, Kumbhare, Dima, Youssef, Zhi Q, Yao, Charles E, McCall, Mohamed El, Gazzar
Publikováno v:
Journal of clinical & cellular immunology
Myeloid-derived suppressor cells (MDSCs) expand during mouse and human sepsis, but the mechanism responsible for this is unclear. We previously reported that nuclear transport of S100A9 protein programs Gr1+CD11b+ myeloid precursors into MDSCs in sep
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::b543be0cf4e5b130950577a0377403da
http://europepmc.org/articles/PMC7744002
http://europepmc.org/articles/PMC7744002
Autor:
Dima Youssef, Gregory A. Hawkin, Isatou Bah, Mohamed El Gazzar, Charles E. McCall, Ajinkya Kumbhare, Zhi Q. Yao, Tuqa Alkhateeb
Publikováno v:
Mol Immunol
Myeloid-derived suppressor cells (MDSCs) contribute to high mortality rates during sepsis, but how sepsis induces MDSCs is unclear. Previously we reported that microRNA (miR)-21 and miR-181b reprogram MDSCs in septic mice by increasing levels of DNA
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a776e19e717c9c8425cd28f5581e8c4d
https://europepmc.org/articles/PMC7519576/
https://europepmc.org/articles/PMC7519576/
Autor:
Isatou Bah, Ajinkya Kumbhare, Mohamed El Gazzar, Zhi Q. Yao, Tuqa Alkhateeb, Dima Youssef, Charles E. McCall
Publikováno v:
Molecular Immunology. 127:78
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::c9dc347044e5337fccbc277f36e30398
https://doi.org/10.1016/j.molimm.2020.08.013
https://doi.org/10.1016/j.molimm.2020.08.013
Autor:
Isatou Bah, Mohamed El Gazzar, Ajinkya Kumbhare, Dima Youssef, Gregory A. Hawkin, Zhi Q. Yao, Tuqa Alkhateeb, Charles E. McCall
Publikováno v:
Molecular Immunology. 127:56
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::e30876bfe1fcd01c8a28e3f774769dd4
https://doi.org/10.1016/j.molimm.2020.08.012
https://doi.org/10.1016/j.molimm.2020.08.012
Publikováno v:
Cell Immunol
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7744d35f2dcb1257662f1bd8d8f0e283
https://doi.org/10.1016/j.cellimm.2020.104218
https://doi.org/10.1016/j.cellimm.2020.104218
Publikováno v:
Critical Care Medicine. 48:12-12
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::d6288a4fddf2d7a80ee1d8148866600d
https://doi.org/10.1097/01.ccm.0000618592.67319.a3
https://doi.org/10.1097/01.ccm.0000618592.67319.a3
The myeloid-related protein S100A9 reprograms Gr1(+)CD11b(+) myeloid precursors into myeloid- derived suppressor cells (MDSCs) during murine sepsis. Here, we show that the immunosuppressive cytokine IL-10 supports S100A9 expression and its nuclear lo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d757186f73d4fdbe3bad25ed0f1bb4c7
https://europepmc.org/articles/PMC6129403/
https://europepmc.org/articles/PMC6129403/
Autor:
Charles E. McCall, Ajinkya Kumbhare, Zhi Q. Yao, Jun Dai, Dima Youssef, Mohamed El Gazzar, Danielle A. Williams
Publikováno v:
Innate immunity
Sepsis-induced immunosuppression increases the risk of chronic infection and reduces survival. Myeloid-derived suppressor cells (MDSCs) expand in the bone marrow and spleen during murine polymicrobial sepsis, contributing to immunosuppression. A bett
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bf8e8a71fd1558b38835b5a7e718ebc3
https://pubmed.ncbi.nlm.nih.gov/29172874
https://pubmed.ncbi.nlm.nih.gov/29172874